Search Page
Save citations to file
Email citations
Send citations to clipboard
Add to Collections
Add to My Bibliography
Create a file for external citation management software
Your saved search
Your RSS Feed
Filters
Results by year
Table representation of search results timeline featuring number of search results per year.
Year | Number of Results |
---|---|
2009 | 1 |
2010 | 1 |
2014 | 1 |
2016 | 1 |
2020 | 1 |
2021 | 1 |
2024 | 0 |
Search Results
4 results
Results by year
Filters applied: . Clear all
Page 1
Polycystin-1 mitigates damage and regulates CTGF expression through AKT activation during cardiac ischemia/reperfusion.
Biochim Biophys Acta Mol Basis Dis. 2021 Jan 1;1867(1):165986. doi: 10.1016/j.bbadis.2020.165986. Epub 2020 Oct 13.
Biochim Biophys Acta Mol Basis Dis. 2021.
PMID: 33065236
Free article.
During ischemia/reperfusion (I/R), cardiomyocytes activate pathways that regulate cell survival and death and release factors that modulate fibroblast-to-myofibroblast differentiation. The mechanisms underlying these effects are not fully understood. Polycystin-1 (PC1) is …
During ischemia/reperfusion (I/R), cardiomyocytes activate pathways that regulate cell survival and death and release factors that modulate …
Activation of Chymotrypsin-Like Activity of the Proteasome during Ischemia Induces Myocardial Dysfunction and Death.
Sanchez G, Berrios D, Olmedo I, Pezoa J, Riquelme JA, Montecinos L, Pedrozo Z, Donoso P.
Sanchez G, et al.
PLoS One. 2016 Aug 16;11(8):e0161068. doi: 10.1371/journal.pone.0161068. eCollection 2016.
PLoS One. 2016.
PMID: 27529620
Free PMC article.
Inhibitors of the ubiquitin-proteasome system improve hemodynamic parameters and decrease the infarct size after ischemia reperfusion. The molecular basis of this protection is not fully understood since most available data report inhibition of the 26 proteasome after isch …
Inhibitors of the ubiquitin-proteasome system improve hemodynamic parameters and decrease the infarct size after ischemia reperfusion. The m …
Item in Clipboard
Calcineurin and its regulator, RCAN1, confer time-of-day changes in susceptibility of the heart to ischemia/reperfusion.
Rotter D, Grinsfelder DB, Parra V, Pedrozo Z, Singh S, Sachan N, Rothermel BA.
Rotter D, et al.
J Mol Cell Cardiol. 2014 Sep;74:103-11. doi: 10.1016/j.yjmcc.2014.05.004. Epub 2014 May 15.
J Mol Cell Cardiol. 2014.
PMID: 24838101
Free PMC article.
In both humans and mice, cardiac damage from ischemia/reperfusion (I/R) is greatest at the transition from sleep to activity. The causes of this window of susceptibility are not fully understood. In the murine heart we have reported high amplitude circadian oscillations in …
In both humans and mice, cardiac damage from ischemia/reperfusion (I/R) is greatest at the transition from sleep to activity. The causes of …
Item in Clipboard
Calpains and proteasomes mediate degradation of ryanodine receptors in a model of cardiac ischemic reperfusion.
Pedrozo Z, Sánchez G, Torrealba N, Valenzuela R, Fernández C, Hidalgo C, Lavandero S, Donoso P.
Pedrozo Z, et al.
Biochim Biophys Acta. 2010 Mar;1802(3):356-62. doi: 10.1016/j.bbadis.2009.12.005. Epub 2009 Dec 21.
Biochim Biophys Acta. 2010.
PMID: 20026269
Free article.
In the heart, ischemia/reperfusion causes a rapid and significant decrease in RyR2 content but the mechanisms responsible for this effect are not fully understood. We have studied the involvement of three proteolytic systems--calpains, the proteasome and autophagy--on the …
In the heart, ischemia/reperfusion causes a rapid and significant decrease in RyR2 content but the mechanisms responsible for this effect ar …
Item in Clipboard
Cite
Cite