Wille CK - Search Results

1

DOT1L interaction partner AF10 controls patterning of H3K79 methylation and RNA polymerase II to maintain cell identity.

Wille CK, Neumann EN, Deshpande AJ, Sridharan R. Wille CK, et al. Stem Cell Reports. 2023 Dec 12;18(12):2451-2463. doi: 10.1016/j.stemcr.2023.10.017. Epub 2023 Nov 22. Stem Cell Reports. 2023. PMID: 37995701 Free PMC article.

2

DOT1L inhibition enhances pluripotency beyond acquisition of epithelial identity and without immediate suppression of the somatic transcriptome.

Wille CK, Sridharan R. Wille CK, et al. Stem Cell Reports. 2022 Feb 8;17(2):384-396. doi: 10.1016/j.stemcr.2021.12.004. Epub 2022 Jan 6. Stem Cell Reports. 2022. PMID: 34995500 Free PMC article.

3

Connecting the DOTs on Cell Identity.

Wille CK, Sridharan R. Wille CK, et al. Front Cell Dev Biol. 2022 Jun 6;10:906713. doi: 10.3389/fcell.2022.906713. eCollection 2022. Front Cell Dev Biol. 2022. PMID: 35733849 Free PMC article. Review.

4

DOT1L is a barrier to histone acetylation during reprogramming to pluripotency.

Wille CK, Zhang X, Haws SA, Denu JM, Sridharan R. Wille CK, et al. Sci Adv. 2023 Nov 15;9(46):eadf3980. doi: 10.1126/sciadv.adf3980. Epub 2023 Nov 17. Sci Adv. 2023. PMID: 37976354 Free PMC article.

5

Methyl-Metabolite Depletion Elicits Adaptive Responses to Support Heterochromatin Stability and Epigenetic Persistence.

Haws SA, Yu D, Ye C, Wille CK, Nguyen LC, Krautkramer KA, Tomasiewicz JL, Yang SE, Miller BR, Liu WH, Igarashi K, Sridharan R, Tu BP, Cryns VL, Lamming DW, Denu JM. Haws SA, et al. Among authors: wille ck. Mol Cell. 2020 Apr 16;78(2):210-223.e8. doi: 10.1016/j.molcel.2020.03.004. Epub 2020 Mar 23. Mol Cell. 2020. PMID: 32208170 Free PMC article.

6

Viral genome methylation differentially affects the ability of BZLF1 versus BRLF1 to activate Epstein-Barr virus lytic gene expression and viral replication.

Wille CK, Nawandar DM, Panfil AR, Ko MM, Hagemeier SR, Kenney SC. Wille CK, et al. J Virol. 2013 Jan;87(2):935-50. doi: 10.1128/JVI.01790-12. Epub 2012 Nov 7. J Virol. 2013. PMID: 23135711 Free PMC article.

7

Restricted TET2 Expression in Germinal Center Type B Cells Promotes Stringent Epstein-Barr Virus Latency.

Wille CK, Li Y, Rui L, Johannsen EC, Kenney SC. Wille CK, et al. J Virol. 2017 Feb 14;91(5):e01987-16. doi: 10.1128/JVI.01987-16. Print 2017 Mar 1. J Virol. 2017. PMID: 28003489 Free PMC article.

8

5-hydroxymethylation of the EBV genome regulates the latent to lytic switch.

Wille CK, Nawandar DM, Henning AN, Ma S, Oetting KM, Lee D, Lambert P, Johannsen EC, Kenney SC. Wille CK, et al. Proc Natl Acad Sci U S A. 2015 Dec 29;112(52):E7257-65. doi: 10.1073/pnas.1513432112. Epub 2015 Dec 9. Proc Natl Acad Sci U S A. 2015. PMID: 26663912 Free PMC article.

9

Cellular transcription factor Oct-1 interacts with the Epstein-Barr virus BRLF1 protein to promote disruption of viral latency.

Robinson AR, Kwek SS, Hagemeier SR, Wille CK, Kenney SC. Robinson AR, et al. Among authors: wille ck. J Virol. 2011 Sep;85(17):8940-53. doi: 10.1128/JVI.00569-11. Epub 2011 Jun 22. J Virol. 2011. PMID: 21697476 Free PMC article.

10

Epstein-Barr Virus Gene BARF1 Expression is Regulated by the Epithelial Differentiation Factor ΔNp63α in Undifferentiated Nasopharyngeal Carcinoma.

Hoebe E, Wille C, Hagemeier S, Kenney S, Greijer A, Middeldorp J. Hoebe E, et al. Cancers (Basel). 2018 Mar 17;10(3):76. doi: 10.3390/cancers10030076. Cancers (Basel). 2018. PMID: 29562599 Free PMC article.

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