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Empagliflozin treatment rescues abnormally reduced Na+ currents in ventricular cardiomyocytes from dystrophin-deficient mdx mice.
Sauer J, Marksteiner J, Lilliu E, Hackl B, Todt H, Kubista H, Dostal C, Podesser BK, Kiss A, Koenig X, Hilber K. Sauer J, et al. Among authors: todt h. Am J Physiol Heart Circ Physiol. 2024 Feb 1;326(2):H418-H425. doi: 10.1152/ajpheart.00729.2023. Epub 2023 Dec 15. Am J Physiol Heart Circ Physiol. 2024. PMID: 38099845 Free article.
Enhanced currents through L-type calcium channels in cardiomyocytes disturb the electrophysiology of the dystrophic heart.
Koenig X, Rubi L, Obermair GJ, Cervenka R, Dang XB, Lukacs P, Kummer S, Bittner RE, Kubista H, Todt H, Hilber K. Koenig X, et al. Among authors: todt h. Am J Physiol Heart Circ Physiol. 2014 Feb 15;306(4):H564-H573. doi: 10.1152/ajpheart.00441.2013. Epub 2013 Dec 13. Am J Physiol Heart Circ Physiol. 2014. PMID: 24337461 Free PMC article.
Neuronal nitric oxide synthase regulation of calcium cycling in ventricular cardiomyocytes is independent of Cav1.2 channel modulation under basal conditions.
Ebner J, Cagalinec M, Kubista H, Todt H, Szabo PL, Kiss A, Podesser BK, Cserne Szappanos H, Hool LC, Hilber K, Koenig X. Ebner J, et al. Among authors: todt h. Pflugers Arch. 2020 Jan;472(1):61-74. doi: 10.1007/s00424-019-02335-7. Epub 2019 Dec 10. Pflugers Arch. 2020. PMID: 31822999 Free PMC article.
136 results