Taira C - Search Results

1

Comparison of molecular structure and fibrin polymerization between two Bβ-chain N-terminal region fibrinogen variants, Bβp.G45C and Bβp.R74C.

Kaido T, Yoda M, Kamijo T, Taira C, Higuchi Y, Okumura N. Kaido T, et al. Among authors: taira c. Int J Hematol. 2020 Sep;112(3):331-340. doi: 10.1007/s12185-020-02919-5. Epub 2020 Jun 19. Int J Hematol. 2020. PMID: 32562089

2

γD318Y fibrinogen shows no fibrin polymerization due to defective "A-a" and "B-b" interactions, whereas that of γK321E fibrinogen is nearly normal.

Kamijo T, Mukai S, Taira C, Higuchi Y, Okumura N. Kamijo T, et al. Among authors: taira c. Thromb Res. 2019 Oct;182:150-158. doi: 10.1016/j.thromres.2019.08.017. Epub 2019 Aug 20. Thromb Res. 2019. PMID: 31484085

3

Heterozygous variant fibrinogen γA289V (Kanazawa III) was confirmed as hypodysfibrinogenemia by plasma and recombinant fibrinogens.

Kaido T, Yoda M, Kamijo T, Taira C, Higuchi Y, Okumura N. Kaido T, et al. Among authors: taira c. Int J Lab Hematol. 2020 Apr;42(2):190-197. doi: 10.1111/ijlh.13152. Epub 2020 Jan 20. Int J Lab Hematol. 2020. PMID: 31957968

4

Congenital fibrinogen disorder with a compound heterozygote possessing two novel FGB mutations, one qualitative and the other quantitative.

Yoda M, Kaido T, Taira C, Higuchi Y, Arai S, Okumura N. Yoda M, et al. Among authors: taira c. Thromb Res. 2020 Dec;196:152-158. doi: 10.1016/j.thromres.2020.08.031. Epub 2020 Aug 20. Thromb Res. 2020. PMID: 32871307

5

A Novel Amino Acid Substitution, Fibrinogen Bβp.Pro234Leu, Associated with Hypofibrinogenemia Causing Impairment of Fibrinogen Assembly and Secretion.

Kaido T, Yoda M, Kamijo T, Arai S, Taira C, Higuchi Y, Okumura N. Kaido T, et al. Among authors: taira c. Int J Mol Sci. 2020 Dec 10;21(24):9422. doi: 10.3390/ijms21249422. Int J Mol Sci. 2020. PMID: 33322044 Free PMC article.

6

Novel variant fibrinogen γp.C352R produced hypodysfibrinogenemia leading to a bleeding episode and failure of infertility treatment.

Yoda M, Kaido T, Kamijo T, Taira C, Higuchi Y, Arai S, Okumura N. Yoda M, et al. Among authors: taira c. Int J Hematol. 2021 Sep;114(3):325-333. doi: 10.1007/s12185-021-03174-y. Epub 2021 Jun 12. Int J Hematol. 2021. PMID: 34117991

7

Fibrin monomers derived from thrombogenic dysfibrinogenemia, Naples-type variant (BβAla68Thr), showed almost entirely normal polymerization.

Kamijo T, Nagata K, Taira C, Higuchi Y, Arai S, Okumura N. Kamijo T, et al. Among authors: taira c. Thromb Res. 2018 Dec;172:1-3. doi: 10.1016/j.thromres.2018.10.004. Epub 2018 Oct 4. Thromb Res. 2018. PMID: 30321757 No abstract available.

8

A novel frameshift mutation in the fibrinogen γC terminal region, FGG c.1169_1170 del AT, leading to hypofibrinogenemia.

Nagata K, Arai S, Taira C, Sugano M, Honda T, Okumura N. Nagata K, et al. Among authors: taira c. Thromb Res. 2017 Nov;159:82-85. doi: 10.1016/j.thromres.2017.10.002. Epub 2017 Oct 2. Thromb Res. 2017. PMID: 28992465 No abstract available.

9

A Novel Mutation in the Fibrinogen Bβ Chain (c.490G>A; End of Exon 3) Causes a Splicing Abnormality and Ultimately Leads to Congenital Hypofibrinogenemia.

Taira C, Matsuda K, Arai S, Sugano M, Uehara T, Okumura N. Taira C, et al. Int J Mol Sci. 2017 Nov 20;18(11):2470. doi: 10.3390/ijms18112470. Int J Mol Sci. 2017. PMID: 29156616 Free PMC article.

10

Application of allele-specific quantitative PCR using genomic DNA to monitor minimal residual disease based on mutant gene levels following allogeneic hematopoietic stem cell transplantation in patients with hematological malignancies: comparison of mutant levels with autologous DNA percentage by short tandem repeat-PCR.

Taira C, Matsuda K, Saito S, Sakashita K, Sugano M, Okumura N, Honda T. Taira C, et al. Clin Chim Acta. 2012 Feb 18;413(3-4):516-9. doi: 10.1016/j.cca.2011.11.017. Epub 2011 Nov 25. Clin Chim Acta. 2012. PMID: 22138486

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