Sudduth TL - Search Results

1

Atorvastatin rescues hyperhomocysteinemia-induced cognitive deficits and neuroinflammatory gene changes.

Weekman EM, Johnson SN, Rogers CB, Sudduth TL, Xie K, Qiao Q, Fardo DW, Bottiglieri T, Wilcock DM. Weekman EM, et al. Among authors: sudduth tl. J Neuroinflammation. 2023 Sep 1;20(1):199. doi: 10.1186/s12974-023-02883-x. J Neuroinflammation. 2023. PMID: 37658433 Free PMC article.

2

Lithium treatment of APPSwDI/NOS2-/- mice leads to reduced hyperphosphorylated tau, increased amyloid deposition and altered inflammatory phenotype.

Sudduth TL, Wilson JG, Everhart A, Colton CA, Wilcock DM. Sudduth TL, et al. PLoS One. 2012;7(2):e31993. doi: 10.1371/journal.pone.0031993. Epub 2012 Feb 9. PLoS One. 2012. PMID: 22347510 Free PMC article.

3

Neuroinflammatory phenotype in early Alzheimer's disease.

Sudduth TL, Schmitt FA, Nelson PT, Wilcock DM. Sudduth TL, et al. Neurobiol Aging. 2013 Apr;34(4):1051-9. doi: 10.1016/j.neurobiolaging.2012.09.012. Epub 2012 Oct 9. Neurobiol Aging. 2013. PMID: 23062700 Free PMC article.

4

Induction of hyperhomocysteinemia models vascular dementia by induction of cerebral microhemorrhages and neuroinflammation.

Sudduth TL, Powell DK, Smith CD, Greenstein A, Wilcock DM. Sudduth TL, et al. J Cereb Blood Flow Metab. 2013 May;33(5):708-15. doi: 10.1038/jcbfm.2013.1. Epub 2013 Jan 30. J Cereb Blood Flow Metab. 2013. PMID: 23361394 Free PMC article.

5

Intracranial injection of Gammagard, a human IVIg, modulates the inflammatory response of the brain and lowers Aβ in APP/PS1 mice along a different time course than anti-Aβ antibodies.

Sudduth TL, Greenstein A, Wilcock DM. Sudduth TL, et al. J Neurosci. 2013 Jun 5;33(23):9684-92. doi: 10.1523/JNEUROSCI.1220-13.2013. J Neurosci. 2013. PMID: 23739965 Free PMC article.

6

β-amyloid deposition is shifted to the vasculature and memory impairment is exacerbated when hyperhomocysteinemia is induced in APP/PS1 transgenic mice.

Sudduth TL, Weekman EM, Brothers HM, Braun K, Wilcock DM. Sudduth TL, et al. Alzheimers Res Ther. 2014 Jun 9;6(3):32. doi: 10.1186/alzrt262. eCollection 2014. Alzheimers Res Ther. 2014. PMID: 24991237 Free PMC article.

7

Transition from an M1 to a mixed neuroinflammatory phenotype increases amyloid deposition in APP/PS1 transgenic mice.

Weekman EM, Sudduth TL, Abner EL, Popa GJ, Mendenhall MD, Brothers HM, Braun K, Greenstein A, Wilcock DM. Weekman EM, et al. Among authors: sudduth tl. J Neuroinflammation. 2014 Jul 25;11:127. doi: 10.1186/1742-2094-11-127. J Neuroinflammation. 2014. PMID: 25062954 Free PMC article.

8

Determining the role of IL-4 induced neuroinflammation in microglial activity and amyloid-β using BV2 microglial cells and APP/PS1 transgenic mice.

Latta CH, Sudduth TL, Weekman EM, Brothers HM, Abner EL, Popa GJ, Mendenhall MD, Gonzalez-Oregon F, Braun K, Wilcock DM. Latta CH, et al. Among authors: sudduth tl. J Neuroinflammation. 2015 Mar 4;12:41. doi: 10.1186/s12974-015-0243-6. J Neuroinflammation. 2015. PMID: 25885682 Free PMC article.

9

Down syndrome individuals with Alzheimer's disease have a distinct neuroinflammatory phenotype compared to sporadic Alzheimer's disease.

Wilcock DM, Hurban J, Helman AM, Sudduth TL, McCarty KL, Beckett TL, Ferrell JC, Murphy MP, Abner EL, Schmitt FA, Head E. Wilcock DM, et al. Among authors: sudduth tl. Neurobiol Aging. 2015 Sep;36(9):2468-74. doi: 10.1016/j.neurobiolaging.2015.05.016. Epub 2015 May 30. Neurobiol Aging. 2015. PMID: 26103884 Free PMC article.

10

Reduced Efficacy of Anti-Aβ Immunotherapy in a Mouse Model of Amyloid Deposition and Vascular Cognitive Impairment Comorbidity.

Weekman EM, Sudduth TL, Caverly CN, Kopper TJ, Phillips OW, Powell DK, Wilcock DM. Weekman EM, et al. Among authors: sudduth tl. J Neurosci. 2016 Sep 21;36(38):9896-907. doi: 10.1523/JNEUROSCI.1762-16.2016. J Neurosci. 2016. PMID: 27656027 Free PMC article.

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