Seemann P - Search Results

1

BMPs in bone regeneration: Less is more effective, a paradigm-shift.

Schmidt-Bleek K, Willie BM, Schwabe P, Seemann P, Duda GN. Schmidt-Bleek K, et al. Among authors: seemann p. Cytokine Growth Factor Rev. 2016 Feb;27:141-8. doi: 10.1016/j.cytogfr.2015.11.006. Epub 2015 Nov 25. Cytokine Growth Factor Rev. 2016. PMID: 26678813 Review.

2

Time kinetics of bone defect healing in response to BMP-2 and GDF-5 characterised by in vivo biomechanics.

Wulsten D, Glatt V, Ellinghaus A, Schmidt-Bleek K, Petersen A, Schell H, Lienau J, Sebald W, Plöger F, Seemann P, Duda GN. Wulsten D, et al. Among authors: seemann p. Eur Cell Mater. 2011 Feb 11;21:177-92. doi: 10.22203/ecm.v021a14. Eur Cell Mater. 2011. PMID: 21312163 Free article.

3

Improved bone defect healing by a superagonistic GDF5 variant derived from a patient with multiple synostoses syndrome.

Degenkolbe E, Schwarz C, Ott CE, König J, Schmidt-Bleek K, Ellinghaus A, Schmidt T, Lienau J, Plöger F, Mundlos S, Duda GN, Willie BM, Seemann P. Degenkolbe E, et al. Among authors: seemann p. Bone. 2015 Apr;73:111-9. doi: 10.1016/j.bone.2014.12.017. Epub 2014 Dec 24. Bone. 2015. PMID: 25543012

4

Treatment with recombinant human bone morphogenetic protein 7 leads to a transient induction of neutralizing autoantibodies in a subset of patients.

Schuette A, Moghaddam A, Seemann P, Duda GN, Schmidmaier G, Schomburg L. Schuette A, et al. Among authors: seemann p. BBA Clin. 2016 Aug 3;6:100-7. doi: 10.1016/j.bbacli.2016.08.001. eCollection 2016 Dec. BBA Clin. 2016. PMID: 27617228 Free PMC article.

5

Functional analysis of alleged NOGGIN mutation G92E disproves its pathogenic relevance.

Zimmer J, Doelken SC, Horn D, Groppe JC, Shore EM, Kaplan FS, Seemann P. Zimmer J, et al. Among authors: seemann p. PLoS One. 2012;7(4):e35062. doi: 10.1371/journal.pone.0035062. Epub 2012 Apr 18. PLoS One. 2012. PMID: 22529972 Free PMC article.

6

Duplications involving a conserved regulatory element downstream of BMP2 are associated with brachydactyly type A2.

Dathe K, Kjaer KW, Brehm A, Meinecke P, Nürnberg P, Neto JC, Brunoni D, Tommerup N, Ott CE, Klopocki E, Seemann P, Mundlos S. Dathe K, et al. Among authors: seemann p. Am J Hum Genet. 2009 Apr;84(4):483-92. doi: 10.1016/j.ajhg.2009.03.001. Epub 2009 Mar 26. Am J Hum Genet. 2009. PMID: 19327734 Free PMC article.

7

Activating and deactivating mutations in the receptor interaction site of GDF5 cause symphalangism or brachydactyly type A2.

Seemann P, Schwappacher R, Kjaer KW, Krakow D, Lehmann K, Dawson K, Stricker S, Pohl J, Plöger F, Staub E, Nickel J, Sebald W, Knaus P, Mundlos S. Seemann P, et al. J Clin Invest. 2005 Sep;115(9):2373-81. doi: 10.1172/JCI25118. Epub 2005 Aug 25. J Clin Invest. 2005. PMID: 16127465 Free PMC article.

8

The Fibrodysplasia Ossificans Progressiva (FOP) mutation p.R206H in ACVR1 confers an altered ligand response.

Hildebrand L, Stange K, Deichsel A, Gossen M, Seemann P. Hildebrand L, et al. Among authors: seemann p. Cell Signal. 2017 Jan;29:23-30. doi: 10.1016/j.cellsig.2016.10.001. Epub 2016 Oct 4. Cell Signal. 2017. PMID: 27713089

9

A novel R486Q mutation in BMPR1B resulting in either a brachydactyly type C/symphalangism-like phenotype or brachydactyly type A2.

Lehmann K, Seemann P, Boergermann J, Morin G, Reif S, Knaus P, Mundlos S. Lehmann K, et al. Among authors: seemann p. Eur J Hum Genet. 2006 Dec;14(12):1248-54. doi: 10.1038/sj.ejhg.5201708. Epub 2006 Sep 6. Eur J Hum Genet. 2006. PMID: 16957682

10

A GDF5 point mutation strikes twice--causing BDA1 and SYNS2.

Degenkolbe E, König J, Zimmer J, Walther M, Reißner C, Nickel J, Plöger F, Raspopovic J, Sharpe J, Dathe K, Hecht JT, Mundlos S, Doelken SC, Seemann P. Degenkolbe E, et al. Among authors: seemann p. PLoS Genet. 2013;9(10):e1003846. doi: 10.1371/journal.pgen.1003846. Epub 2013 Oct 3. PLoS Genet. 2013. PMID: 24098149 Free PMC article.

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