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CLCN2 chloride channel mutations in familial hyperaldosteronism type II.
Scholl UI, Stölting G, Schewe J, Thiel A, Tan H, Nelson-Williams C, Vichot AA, Jin SC, Loring E, Untiet V, Yoo T, Choi J, Xu S, Wu A, Kirchner M, Mertins P, Rump LC, Onder AM, Gamble C, McKenney D, Lash RW, Jones DP, Chune G, Gagliardi P, Choi M, Gordon R, Stowasser M, Fahlke C, Lifton RP. Scholl UI, et al. Among authors: schewe j. Nat Genet. 2018 Mar;50(3):349-354. doi: 10.1038/s41588-018-0048-5. Epub 2018 Feb 5. Nat Genet. 2018. PMID: 29403011 Free PMC article.
Genetic causes of primary aldosteronism.
Seidel E, Schewe J, Scholl UI. Seidel E, et al. Among authors: schewe j. Exp Mol Med. 2019 Nov 6;51(11):1-12. doi: 10.1038/s12276-019-0337-9. Exp Mol Med. 2019. PMID: 31695023 Free PMC article. Review.
Enhanced Ca2+ signaling, mild primary aldosteronism, and hypertension in a familial hyperaldosteronism mouse model (Cacna1hM1560V/+ ).
Seidel E, Schewe J, Zhang J, Dinh HA, Forslund SK, Markó L, Hellmig N, Peters J, Muller DN, Lifton RP, Nottoli T, Stölting G, Scholl UI. Seidel E, et al. Among authors: schewe j. Proc Natl Acad Sci U S A. 2021 Apr 27;118(17):e2014876118. doi: 10.1073/pnas.2014876118. Proc Natl Acad Sci U S A. 2021. PMID: 33879608 Free PMC article.
121 results