Pieraccioli M - Search Results

1

Dual inhibition of CDK12 and CDK13 uncovers actionable vulnerabilities in patient-derived ovarian cancer organoids.

Cesari E, Ciucci A, Pieraccioli M, Caggiano C, Nero C, Bonvissuto D, Sillano F, Buttarelli M, Piermattei A, Loverro M, Camarda F, Greco V, De Bonis M, Minucci A, Gallo D, Urbani A, Vizzielli G, Scambia G, Sette C. Cesari E, et al. Among authors: pieraccioli m. J Exp Clin Cancer Res. 2023 May 18;42(1):126. doi: 10.1186/s13046-023-02682-5. J Exp Clin Cancer Res. 2023. PMID: 37202753 Free PMC article.

2

ZNF281 inhibits neuronal differentiation and is a prognostic marker for neuroblastoma.

Pieraccioli M, Nicolai S, Pitolli C, Agostini M, Antonov A, Malewicz M, Knight RA, Raschellà G, Melino G. Pieraccioli M, et al. Proc Natl Acad Sci U S A. 2018 Jul 10;115(28):7356-7361. doi: 10.1073/pnas.1801435115. Epub 2018 Jun 25. Proc Natl Acad Sci U S A. 2018. PMID: 29941555 Free PMC article.

3

The Splicing Factor PTBP1 Promotes Expression of Oncogenic Splice Variants and Predicts Poor Prognosis in Patients with Non-muscle-Invasive Bladder Cancer.

Bielli P, Panzeri V, Lattanzio R, Mutascio S, Pieraccioli M, Volpe E, Pagliarulo V, Piantelli M, Giannantoni A, Di Stasi SM, Sette C. Bielli P, et al. Among authors: pieraccioli m. Clin Cancer Res. 2018 Nov 1;24(21):5422-5432. doi: 10.1158/1078-0432.CCR-17-3850. Epub 2018 Jul 16. Clin Cancer Res. 2018. PMID: 30012566

4

c-MYC empowers transcription and productive splicing of the oncogenic splicing factor Sam68 in cancer.

Caggiano C, Pieraccioli M, Panzeri V, Sette C, Bielli P. Caggiano C, et al. Among authors: pieraccioli m. Nucleic Acids Res. 2019 Jul 9;47(12):6160-6171. doi: 10.1093/nar/gkz344. Nucleic Acids Res. 2019. PMID: 31066450 Free PMC article.

5

Splicing Dysregulation as Oncogenic Driver and Passenger Factor in Brain Tumors.

Bielli P, Pagliarini V, Pieraccioli M, Caggiano C, Sette C. Bielli P, et al. Among authors: pieraccioli m. Cells. 2019 Dec 18;9(1):10. doi: 10.3390/cells9010010. Cells. 2019. PMID: 31861467 Free PMC article. Review.

6

Combinatorial control of Spo11 alternative splicing by modulation of RNA polymerase II dynamics and splicing factor recruitment during meiosis.

Cesari E, Loiarro M, Naro C, Pieraccioli M, Farini D, Pellegrini L, Pagliarini V, Bielli P, Sette C. Cesari E, et al. Among authors: pieraccioli m. Cell Death Dis. 2020 Apr 17;11(4):240. doi: 10.1038/s41419-020-2443-y. Cell Death Dis. 2020. PMID: 32303676 Free PMC article.

7

The DNA/RNA helicase DHX9 contributes to the transcriptional program of the androgen receptor in prostate cancer.

Chellini L, Pieraccioli M, Sette C, Paronetto MP. Chellini L, et al. Among authors: pieraccioli m. J Exp Clin Cancer Res. 2022 May 19;41(1):178. doi: 10.1186/s13046-022-02384-4. J Exp Clin Cancer Res. 2022. PMID: 35590370 Free PMC article.

8

The androgen receptor couples promoter recruitment of RNA processing factors to regulation of alternative polyadenylation at the 3' end of transcripts.

Caggiano C, Pieraccioli M, Pitolli C, Babini G, Zheng D, Tian B, Bielli P, Sette C. Caggiano C, et al. Among authors: pieraccioli m. Nucleic Acids Res. 2022 Sep 23;50(17):9780-9796. doi: 10.1093/nar/gkac737. Nucleic Acids Res. 2022. PMID: 36043441 Free PMC article.

9

The transcriptional terminator XRN2 and the RNA-binding protein Sam68 link alternative polyadenylation to cell cycle progression in prostate cancer.

Pieraccioli M, Caggiano C, Mignini L, Zhong C, Babini G, Lattanzio R, Di Stasi S, Tian B, Sette C, Bielli P. Pieraccioli M, et al. Nat Struct Mol Biol. 2022 Nov;29(11):1101-1112. doi: 10.1038/s41594-022-00853-0. Epub 2022 Nov 7. Nat Struct Mol Biol. 2022. PMID: 36344846 Free PMC article.

10

CDK12/13 promote splicing of proximal introns by enhancing the interaction between RNA polymerase II and the splicing factor SF3B1.

Panzeri V, Pieraccioli M, Cesari E, de la Grange P, Sette C. Panzeri V, et al. Among authors: pieraccioli m. Nucleic Acids Res. 2023 Jun 23;51(11):5512-5526. doi: 10.1093/nar/gkad258. Nucleic Acids Res. 2023. PMID: 37026485 Free PMC article.

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