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Early activation of matrix metalloproteinases underlies the exacerbated systolic and diastolic dysfunction in mice lacking TIMP3 following myocardial infarction.
Kandalam V, Basu R, Abraham T, Wang X, Awad A, Wang W, Lopaschuk GD, Maeda N, Oudit GY, Kassiri Z. Kandalam V, et al. Among authors: oudit gy. Am J Physiol Heart Circ Physiol. 2010 Oct;299(4):H1012-23. doi: 10.1152/ajpheart.00246.2010. Epub 2010 Jul 30. Am J Physiol Heart Circ Physiol. 2010. PMID: 20675565 Free PMC article.
Myocardial ATGL overexpression decreases the reliance on fatty acid oxidation and protects against pressure overload-induced cardiac dysfunction.
Kienesberger PC, Pulinilkunnil T, Sung MM, Nagendran J, Haemmerle G, Kershaw EE, Young ME, Light PE, Oudit GY, Zechner R, Dyck JR. Kienesberger PC, et al. Among authors: oudit gy. Mol Cell Biol. 2012 Feb;32(4):740-50. doi: 10.1128/MCB.06470-11. Epub 2011 Dec 12. Mol Cell Biol. 2012. PMID: 22158969 Free PMC article.
Agonist-induced hypertrophy and diastolic dysfunction are associated with selective reduction in glucose oxidation: a metabolic contribution to heart failure with normal ejection fraction.
Mori J, Basu R, McLean BA, Das SK, Zhang L, Patel VB, Wagg CS, Kassiri Z, Lopaschuk GD, Oudit GY. Mori J, et al. Among authors: oudit gy. Circ Heart Fail. 2012 Jul 1;5(4):493-503. doi: 10.1161/CIRCHEARTFAILURE.112.966705. Epub 2012 Jun 15. Circ Heart Fail. 2012. PMID: 22705769
344 results