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p53 amyloid formation leading to its loss of function: implications in cancer pathogenesis.
Ghosh S, Salot S, Sengupta S, Navalkar A, Ghosh D, Jacob R, Das S, Kumar R, Jha NN, Sahay S, Mehra S, Mohite GM, Ghosh SK, Kombrabail M, Krishnamoorthy G, Chaudhari P, Maji SK. Ghosh S, et al. Among authors: maji sk. Cell Death Differ. 2017 Oct;24(10):1784-1798. doi: 10.1038/cdd.2017.105. Epub 2017 Jun 23. Cell Death Differ. 2017. PMID: 28644435 Free PMC article.
Comparison of Kinetics, Toxicity, Oligomer Formation, and Membrane Binding Capacity of α-Synuclein Familial Mutations at the A53 Site, Including the Newly Discovered A53V Mutation.
Mohite GM, Kumar R, Panigrahi R, Navalkar A, Singh N, Datta D, Mehra S, Ray S, Gadhe LG, Das S, Singh N, Chatterjee D, Kumar A, Maji SK. Mohite GM, et al. Among authors: maji sk. Biochemistry. 2018 Sep 4;57(35):5183-5187. doi: 10.1021/acs.biochem.8b00314. Epub 2018 May 21. Biochemistry. 2018. PMID: 29771508
Glycosaminoglycans have variable effects on α-synuclein aggregation and differentially affect the activities of the resulting amyloid fibrils.
Mehra S, Ghosh D, Kumar R, Mondal M, Gadhe LG, Das S, Anoop A, Jha NN, Jacob RS, Chatterjee D, Ray S, Singh N, Kumar A, Maji SK. Mehra S, et al. Among authors: maji sk. J Biol Chem. 2018 Aug 24;293(34):12975-12991. doi: 10.1074/jbc.RA118.004267. Epub 2018 Jun 29. J Biol Chem. 2018. PMID: 29959225 Free PMC article.
135 results