Kim SG - Search Results

1

Synthetic lethality by targeting the RUVBL1/2-TTT complex in mTORC1-hyperactive cancer cells.

Shin SH, Lee JS, Zhang JM, Choi S, Boskovic ZV, Zhao R, Song M, Wang R, Tian J, Lee MH, Kim JH, Jeong M, Lee JH, Petukhov M, Lee SW, Kim SG, Zou L, Byun S. Shin SH, et al. Among authors: kim sg, kim jh. Sci Adv. 2020 Jul 31;6(31):eaay9131. doi: 10.1126/sciadv.aay9131. eCollection 2020 Jul. Sci Adv. 2020. PMID: 32789167 Free PMC article.

2

Metabolic stress controls mTORC1 lysosomal localization and dimerization by regulating the TTT-RUVBL1/2 complex.

Kim SG, Hoffman GR, Poulogiannis G, Buel GR, Jang YJ, Lee KW, Kim BY, Erikson RL, Cantley LC, Choo AY, Blenis J. Kim SG, et al. Among authors: kim by. Mol Cell. 2013 Jan 10;49(1):172-85. doi: 10.1016/j.molcel.2012.10.003. Epub 2012 Nov 8. Mol Cell. 2013. PMID: 23142078 Free PMC article.

3

Nutrient regulation of the mTOR complex 1 signaling pathway.

Kim SG, Buel GR, Blenis J. Kim SG, et al. Mol Cells. 2013 Jun;35(6):463-73. doi: 10.1007/s10059-013-0138-2. Epub 2013 May 20. Mol Cells. 2013. PMID: 23694989 Free PMC article. Review.

4

Rap1-GTPases control mTORC1 activity by coordinating lysosome organization with amino acid availability.

Mutvei AP, Nagiec MJ, Hamann JC, Kim SG, Vincent CT, Blenis J. Mutvei AP, et al. Among authors: kim sg. Nat Commun. 2020 Mar 17;11(1):1416. doi: 10.1038/s41467-020-15156-5. Nat Commun. 2020. PMID: 32184389 Free PMC article.

5

Glucose addiction of TSC null cells is caused by failed mTORC1-dependent balancing of metabolic demand with supply.

Choo AY, Kim SG, Vander Heiden MG, Mahoney SJ, Vu H, Yoon SO, Cantley LC, Blenis J. Choo AY, et al. Among authors: kim sg. Mol Cell. 2010 May 28;38(4):487-99. doi: 10.1016/j.molcel.2010.05.007. Mol Cell. 2010. PMID: 20513425 Free PMC article.

6

mTORC1 signaling aids in CADalyzing pyrimidine biosynthesis.

Buel GR, Kim SG, Blenis J. Buel GR, et al. Among authors: kim sg. Cell Metab. 2013 May 7;17(5):633-5. doi: 10.1016/j.cmet.2013.04.018. Cell Metab. 2013. PMID: 23663734 Free article.

7

ATM: Promoter of metabolic "cost" reduction and "savings" usage during hypoxia through mTORC1 regulation.

Kim SG, Choo AY, Blenis J. Kim SG, et al. Mol Cell. 2010 Nov 24;40(4):501-2. doi: 10.1016/j.molcel.2010.11.013. Mol Cell. 2010. PMID: 21095578 Free article.

8

Rapamycin differentially inhibits S6Ks and 4E-BP1 to mediate cell-type-specific repression of mRNA translation.

Choo AY, Yoon SO, Kim SG, Roux PP, Blenis J. Choo AY, et al. Among authors: kim sg. Proc Natl Acad Sci U S A. 2008 Nov 11;105(45):17414-9. doi: 10.1073/pnas.0809136105. Epub 2008 Oct 27. Proc Natl Acad Sci U S A. 2008. PMID: 18955708 Free PMC article.

9

Rapamycin: one drug, many effects.

Li J, Kim SG, Blenis J. Li J, et al. Among authors: kim sg. Cell Metab. 2014 Mar 4;19(3):373-9. doi: 10.1016/j.cmet.2014.01.001. Epub 2014 Feb 6. Cell Metab. 2014. PMID: 24508508 Free PMC article. Review.

10

Editorial Note to: Glucose Addiction of TSC Null Cells Is Caused by Failed mTORC1-Dependent Balancing of Metabolic Demand with Supply.

Choo AY, Kim SG, Vander Heiden MG, Mahoney SJ, Vu H, Yoon SO, Cantley LC, Blenis J. Choo AY, et al. Among authors: kim sg. Mol Cell. 2021 Jul 15;81(14):3031. doi: 10.1016/j.molcel.2021.06.029. Mol Cell. 2021. PMID: 34270944 Free article. No abstract available.

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