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Genetic deletion of Cav3.2 T-type calcium channels abolishes H2S-dependent somatic and visceral pain signaling in C57BL/6 mice.
Matsui K, Tsubota M, Fukushi S, Koike N, Masuda H, Kasanami Y, Miyazaki T, Sekiguchi F, Ohkubo T, Yoshida S, Mukai Y, Oita A, Takada M, Kawabata A. Matsui K, et al. Among authors: kasanami y. J Pharmacol Sci. 2019 Jul;140(3):310-312. doi: 10.1016/j.jphs.2019.07.010. Epub 2019 Jul 30. J Pharmacol Sci. 2019. PMID: 31492577 Free article.
Discovery of pimozide derivatives as novel T-type calcium channel inhibitors with little binding affinity to dopamine D2 receptors for treatment of somatic and visceral pain.
Kasanami Y, Ishikawa C, Kino T, Chonan M, Toyooka N, Takashima Y, Iba Y, Sekiguchi F, Tsubota M, Ohkubo T, Yoshida S, Kawase A, Okada T, Kawabata A. Kasanami Y, et al. Eur J Med Chem. 2022 Dec 5;243:114716. doi: 10.1016/j.ejmech.2022.114716. Epub 2022 Aug 27. Eur J Med Chem. 2022. PMID: 36075145
A hydrolysate of poly-trans-[(2-carboxyethyl)germasesquioxane] (Ge-132) suppresses Cav3.2-dependent pain by sequestering exogenous and endogenous sulfide.
Sekiguchi F, Koike N, Shimada Y, Sugimoto K, Masuda H, Nakamura T, Yamaguchi H, Tanabe G, Marumoto S, Kasanami Y, Tsubota M, Ohkubo T, Yoshida S, Kawabata A. Sekiguchi F, et al. Among authors: kasanami y. Redox Biol. 2023 Feb;59:102579. doi: 10.1016/j.redox.2022.102579. Epub 2022 Dec 15. Redox Biol. 2023. PMID: 36563535 Free PMC article.