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Interleukin-36γ Is Elevated in Cervicovaginal Epithelial Cells in Women With Bacterial Vaginosis and In Vitro After Infection With Microbes Associated With Bacterial Vaginosis.
J Infect Dis. 2020 Mar 2;221(6):983-988. doi: 10.1093/infdis/jiz514.
J Infect Dis. 2020.
PMID: 31586390
Free PMC article.
IL-36γ Is a Key Regulator of Neutrophil Infiltration in the Vaginal Microenvironment and Limits Neuroinvasion in Genital HSV-2 Infection.
Gardner JK, Swaims-Kohlmeier A, Herbst-Kralovetz MM.
Gardner JK, et al.
J Immunol. 2019 Nov 15;203(10):2655-2664. doi: 10.4049/jimmunol.1900280. Epub 2019 Oct 2.
J Immunol. 2019.
PMID: 31578266
Free PMC article.
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IL-36γ induces a transient HSV-2 resistant environment that protects against genital disease and pathogenesis.
Gardner JK, Herbst-Kralovetz MM.
Gardner JK, et al.
Cytokine. 2018 Nov;111:63-71. doi: 10.1016/j.cyto.2018.07.034. Epub 2018 Aug 15.
Cytokine. 2018.
PMID: 30118914
Free PMC article.
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Three-Dimensional Rotating Wall Vessel-Derived Cell Culture Models for Studying Virus-Host Interactions.
Gardner JK, Herbst-Kralovetz MM.
Gardner JK, et al.
Viruses. 2016 Nov 9;8(11):304. doi: 10.3390/v8110304.
Viruses. 2016.
PMID: 27834891
Free PMC article.
Review.
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