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Genetic Complementation of ATP Synthase Deficiency Due to Dysfunction of TMEM70 Assembly Factor in Rat.
Marković A, Tauchmannová K, Šimáková M, Mlejnek P, Kaplanová V, Pecina P, Pecinová A, Papoušek F, Liška F, Šilhavý J, Mikešová J, Neckář J, Houštěk J, Pravenec M, Mráček T. Marković A, et al. Among authors: houstek j. Biomedicines. 2022 Jan 26;10(2):276. doi: 10.3390/biomedicines10020276. Biomedicines. 2022. PMID: 35203486 Free PMC article.
Mitochondrial diseases and genetic defects of ATP synthase.
Houstek J, Pícková A, Vojtísková A, Mrácek T, Pecina P, Jesina P. Houstek J, et al. Biochim Biophys Acta. 2006 Sep-Oct;1757(9-10):1400-5. doi: 10.1016/j.bbabio.2006.04.006. Epub 2006 Apr 19. Biochim Biophys Acta. 2006. PMID: 16730639 Free article. Review.
TMEM70 facilitates biogenesis of mammalian ATP synthase by promoting subunit c incorporation into the rotor structure of the enzyme.
Kovalčíkova J, Vrbacký M, Pecina P, Tauchmannová K, Nůsková H, Kaplanová V, Brázdová A, Alán L, Eliáš J, Čunátová K, Kořínek V, Sedlacek R, Mráček T, Houštěk J. Kovalčíkova J, et al. Among authors: houstek j. FASEB J. 2019 Dec;33(12):14103-14117. doi: 10.1096/fj.201900685RR. Epub 2019 Oct 25. FASEB J. 2019. PMID: 31652072
The biologic role of TMEM70 is to increase the low efficacy of spontaneous assembly of subunit c oligomer, the key and rate-limiting step of ATP-synthase biogenesis, and thus to reach an adequately high physiologic level of ATP synthase in mammalian tissues.-Kovalcikova, J …
The biologic role of TMEM70 is to increase the low efficacy of spontaneous assembly of subunit c oligomer, the key and rate-limiting step of …
Nuclear genetic defects of mitochondrial ATP synthase.
Hejzlarová K, Mráček T, Vrbacký M, Kaplanová V, Karbanová V, Nůsková H, Pecina P, Houštěk J. Hejzlarová K, et al. Among authors: houstek j. Physiol Res. 2014;63(Suppl 1):S57-71. doi: 10.33549/physiolres.932643. Physiol Res. 2014. PMID: 24564666 Free article. Review.
Conplastic strains for identification of retrograde effects of mitochondrial dna variation on cardiometabolic traits in the spontaneously hypertensive rat.
Pravenec M, Šilhavý J, Mlejnek P, Šimáková M, Mráček T, Pecinová A, Tauchmannová K, Hütl M, Malínská H, Kazdová L, Neckář J, Kolář F, Žurmanová J, Novotný J, Houštěk J. Pravenec M, et al. Among authors: houstek j. Physiol Res. 2021 Dec 30;70(Suppl4):S471-S484. doi: 10.33549/physiolres.934740. Physiol Res. 2021. PMID: 35199537 Free PMC article. Review.
CD36 overexpression predisposes to arrhythmias but reduces infarct size in spontaneously hypertensive rats: gene expression profile analysis.
Neckář J, Šilhavy J, Zídek V, Landa V, Mlejnek P, Šimáková M, Seidman JG, Seidman C, Kazdová L, Klevstig M, Novák F, Vecka M, Papoušek F, Houštěk J, Drahota Z, Kurtz TW, Kolář F, Pravenec M. Neckář J, et al. Among authors: houstek j. Physiol Genomics. 2012 Feb 1;44(2):173-82. doi: 10.1152/physiolgenomics.00083.2011. Epub 2011 Nov 29. Physiol Genomics. 2012. PMID: 22128087 Free PMC article.
416 results