Helwani FM - Search Results

1

Prostacyclin is an endosteal bone marrow niche component and its clinical analog iloprost protects hematopoietic stem cell potential during stress.

Tay J, Barbier V, Helwani FM, Price GR, Levesque JP, Winkler IG. Tay J, et al. Among authors: helwani fm. Stem Cells. 2021 Nov;39(11):1532-1545. doi: 10.1002/stem.3438. Stem Cells. 2021. PMID: 34260805

2

The endosteal 'osteoblastic' niche and its role in hematopoietic stem cell homing and mobilization.

Lévesque JP, Helwani FM, Winkler IG. Lévesque JP, et al. Among authors: helwani fm. Leukemia. 2010 Dec;24(12):1979-92. doi: 10.1038/leu.2010.214. Epub 2010 Sep 23. Leukemia. 2010. PMID: 20861913 Review.

3

Hematopoietic progenitor cell mobilization results in hypoxia with increased hypoxia-inducible transcription factor-1 alpha and vascular endothelial growth factor A in bone marrow.

Lévesque JP, Winkler IG, Hendy J, Williams B, Helwani F, Barbier V, Nowlan B, Nilsson SK. Lévesque JP, et al. Stem Cells. 2007 Aug;25(8):1954-65. doi: 10.1634/stemcells.2006-0688. Epub 2007 May 3. Stem Cells. 2007. PMID: 17478585

4

Bone marrow macrophages maintain hematopoietic stem cell (HSC) niches and their depletion mobilizes HSCs.

Winkler IG, Sims NA, Pettit AR, Barbier V, Nowlan B, Helwani F, Poulton IJ, van Rooijen N, Alexander KA, Raggatt LJ, Lévesque JP. Winkler IG, et al. Blood. 2010 Dec 2;116(23):4815-28. doi: 10.1182/blood-2009-11-253534. Epub 2010 Aug 16. Blood. 2010. PMID: 20713966 Free article.

5

Hypoxia inducible factor (HIF)-2α accelerates disease progression in mouse models of leukemia and lymphoma but is not a poor prognosis factor in human AML.

Forristal CE, Brown AL, Helwani FM, Winkler IG, Nowlan B, Barbier V, Powell RJ, Engler GA, Diakiw SM, Zannettino AC, Martin S, Pattabiraman D, D'Andrea RJ, Lewis ID, Levesque JP. Forristal CE, et al. Among authors: helwani fm. Leukemia. 2015 Oct;29(10):2075-85. doi: 10.1038/leu.2015.102. Epub 2015 Apr 29. Leukemia. 2015. PMID: 25921247

6

B-lymphopoiesis is stopped by mobilizing doses of G-CSF and is rescued by overexpression of the anti-apoptotic protein Bcl2.

Winkler IG, Bendall LJ, Forristal CE, Helwani F, Nowlan B, Barbier V, Shen Y, Cisterne A, Sedger LM, Levesque JP. Winkler IG, et al. Haematologica. 2013 Mar;98(3):325-33. doi: 10.3324/haematol.2012.069260. Epub 2012 Aug 28. Haematologica. 2013. PMID: 22929978 Free PMC article.

7

Cortactin is a functional target of E-cadherin-activated Src family kinases in MCF7 epithelial monolayers.

Ren G, Helwani FM, Verma S, McLachlan RW, Weed SA, Yap AS. Ren G, et al. Among authors: helwani fm. J Biol Chem. 2009 Jul 10;284(28):18913-22. doi: 10.1074/jbc.M109.000307. Epub 2009 May 19. J Biol Chem. 2009. PMID: 19457864 Free PMC article.

8

Arp2/3 activity is necessary for efficient formation of E-cadherin adhesive contacts.

Verma S, Shewan AM, Scott JA, Helwani FM, den Elzen NR, Miki H, Takenawa T, Yap AS. Verma S, et al. Among authors: helwani fm. J Biol Chem. 2004 Aug 6;279(32):34062-70. doi: 10.1074/jbc.M404814200. Epub 2004 May 24. J Biol Chem. 2004. PMID: 15159390 Free article.

9

E-cadherin adhesion activates c-Src signaling at cell-cell contacts.

McLachlan RW, Kraemer A, Helwani FM, Kovacs EM, Yap AS. McLachlan RW, et al. Among authors: helwani fm. Mol Biol Cell. 2007 Aug;18(8):3214-23. doi: 10.1091/mbc.e06-12-1154. Epub 2007 Jun 6. Mol Biol Cell. 2007. PMID: 17553930 Free PMC article.

10

Cortactin is necessary for E-cadherin-mediated contact formation and actin reorganization.

Helwani FM, Kovacs EM, Paterson AD, Verma S, Ali RG, Fanning AS, Weed SA, Yap AS. Helwani FM, et al. J Cell Biol. 2004 Mar 15;164(6):899-910. doi: 10.1083/jcb.200309034. J Cell Biol. 2004. PMID: 15024035 Free PMC article.

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