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Abolishing β-arrestin recruitment is necessary for the full metabolic benefits of G protein-biased glucagon-like peptide-1 receptor agonists.
Diabetes Obes Metab. 2024 Jan;26(1):65-77. doi: 10.1111/dom.15288. Epub 2023 Oct 5.
Diabetes Obes Metab. 2024.
PMID: 37795639
Divergent acute versus prolonged pharmacological GLP-1R responses in adult β cell-specific β-arrestin 2 knockout mice.
Bitsi S, El Eid L, Manchanda Y, Oqua AI, Mohamed N, Hansen B, Suba K, Rutter GA, Salem V, Jones B, Tomas A.
Bitsi S, et al. Among authors: el eid l.
Sci Adv. 2023 May 3;9(18):eadf7737. doi: 10.1126/sciadv.adf7737. Epub 2023 May 3.
Sci Adv. 2023.
PMID: 37134170
Free PMC article.
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Control of human pancreatic beta cell kinome by glucagon-like peptide-1 receptor biased agonism.
Xiao J, El Eid L, Buenaventura T, Boutry R, Bonnefond A, Jones B, Rutter GA, Froguel P, Tomas A.
Xiao J, et al. Among authors: el eid l.
Diabetes Obes Metab. 2023 Aug;25(8):2105-2119. doi: 10.1111/dom.15083. Epub 2023 Apr 25.
Diabetes Obes Metab. 2023.
PMID: 37039251
Free PMC article.
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Biased agonism and polymorphic variation at the GLP-1 receptor: Implications for the development of personalised therapeutics.
El Eid L, Reynolds CA, Tomas A, Ben Jones.
El Eid L, et al.
Pharmacol Res. 2022 Oct;184:106411. doi: 10.1016/j.phrs.2022.106411. Epub 2022 Aug 22.
Pharmacol Res. 2022.
PMID: 36007775
Free article.
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