Boidot R - Search Results

1

Sirtuin-1 Activation Controls Tumor Growth by Impeding Th17 Differentiation via STAT3 Deacetylation.

Limagne E, Thibaudin M, Euvrard R, Berger H, Chalons P, Végan F, Humblin E, Boidot R, Rébé C, Derangère V, Ladoire S, Apetoh L, Delmas D, Ghiringhelli F. Limagne E, et al. Among authors: boidot r. Cell Rep. 2017 Apr 25;19(4):746-759. doi: 10.1016/j.celrep.2017.04.004. Cell Rep. 2017. PMID: 28445726 Free article.

2

FOXP3 expression in cancer cells and anthracyclines efficacy in patients with primary breast cancer treated with adjuvant chemotherapy in the phase III UNICANCER-PACS 01 trial.

Ladoire S, Mignot G, Dalban C, Chevriaux A, Arnould L, Rébé C, Apetoh L, Boidot R, Penault-Llorca F, Fumoleau P, Roché H, Spielmann M, Levy C, Lortholary A, Eichler F, Mesleard C, Bonnetain F, Ghiringhelli F. Ladoire S, et al. Among authors: boidot r. Ann Oncol. 2012 Oct;23(10):2552-2561. doi: 10.1093/annonc/mds028. Epub 2012 Mar 19. Ann Oncol. 2012. PMID: 22431701 Free article. Clinical Trial.

3

The transcription factor IRF1 dictates the IL-21-dependent anticancer functions of TH9 cells.

Végran F, Berger H, Boidot R, Mignot G, Bruchard M, Dosset M, Chalmin F, Rébé C, Dérangère V, Ryffel B, Kato M, Prévost-Blondel A, Ghiringhelli F, Apetoh L. Végran F, et al. Among authors: boidot r. Nat Immunol. 2014 Aug;15(8):758-66. doi: 10.1038/ni.2925. Epub 2014 Jun 29. Nat Immunol. 2014. PMID: 24973819

4

Next-generation sequencing analysis of lung and colon carcinomas reveals a variety of genetic alterations.

Chevrier S, Arnould L, Ghiringhelli F, Coudert B, Fumoleau P, Boidot R. Chevrier S, et al. Among authors: boidot r. Int J Oncol. 2014 Sep;45(3):1167-74. doi: 10.3892/ijo.2014.2528. Epub 2014 Jun 27. Int J Oncol. 2014. PMID: 24990411

5

The role of telomeres in predicting individual radiosensitivity of patients with cancer in the era of personalized radiotherapy.

Mirjolet C, Boidot R, Saliques S, Ghiringhelli F, Maingon P, Créhange G. Mirjolet C, et al. Among authors: boidot r. Cancer Treat Rev. 2015 Apr;41(4):354-60. doi: 10.1016/j.ctrv.2015.02.005. Epub 2015 Feb 11. Cancer Treat Rev. 2015. PMID: 25704912 Review.

6

The receptor NLRP3 is a transcriptional regulator of TH2 differentiation.

Bruchard M, Rebé C, Derangère V, Togbé D, Ryffel B, Boidot R, Humblin E, Hamman A, Chalmin F, Berger H, Chevriaux A, Limagne E, Apetoh L, Végran F, Ghiringhelli F. Bruchard M, et al. Among authors: boidot r. Nat Immunol. 2015 Aug;16(8):859-70. doi: 10.1038/ni.3202. Epub 2015 Jun 22. Nat Immunol. 2015. PMID: 26098997

7

Liver X receptor ligand cytotoxicity in colon cancer cells and not in normal colon epithelial cells depends on LXRβ subcellular localization.

Courtaut F, Derangère V, Chevriaux A, Ladoire S, Cotte AK, Arnould L, Boidot R, Rialland M, Ghiringhelli F, Rébé C. Courtaut F, et al. Among authors: boidot r. Oncotarget. 2015 Sep 29;6(29):26651-62. doi: 10.18632/oncotarget.5791. Oncotarget. 2015. PMID: 26450852 Free PMC article.

8

Transcriptome analysis of TH2 CD4(+) T cells differentiated from wild-type and NLRP3KO mice.

Bruchard M, Boidot R, Ghiringhelli F, Végran F. Bruchard M, et al. Among authors: boidot r. Genom Data. 2015 Jul 9;5:314-5. doi: 10.1016/j.gdata.2015.06.031. eCollection 2015 Sep. Genom Data. 2015. PMID: 26484275 Free PMC article.

9

HRAS G13D, a new mutation implicated in the resistance to anti-EGFR therapies in colorectal cancer, a case report.

Boidot R, Chevrier S, Julie V, Ladoire S, Ghiringhelli F. Boidot R, et al. Int J Colorectal Dis. 2016 Jun;31(6):1245-6. doi: 10.1007/s00384-015-2448-7. Epub 2015 Nov 12. Int J Colorectal Dis. 2016. PMID: 26561417 No abstract available.

10

Corrigendum: The receptor NLRP3 is a transcriptional regulator of TH2 differentiation.

Bruchard M, Rebé C, Derangère V, Togbé D, Ryffel B, Boidot R, Humblin E, Hamman A, Chalmin F, Berger H, Chevriaux A, Limagne E, Apetoh L, Végran F, Ghiringhelli F. Bruchard M, et al. Among authors: boidot r. Nat Immunol. 2015 Dec;16(12):1292. doi: 10.1038/ni1215-1292a. Nat Immunol. 2015. PMID: 26580508 No abstract available.

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