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Major basic protein-1 promotes fibrosis of dystrophic muscle and attenuates the cellular immune response in muscular dystrophy.
Hum Mol Genet. 2008 Aug 1;17(15):2280-92. doi: 10.1093/hmg/ddn129. Epub 2008 Apr 21.
Hum Mol Genet. 2008.
PMID: 18430716
Free PMC article.
Age-related loss of nitric oxide synthase in skeletal muscle causes reductions in calpain S-nitrosylation that increase myofibril degradation and sarcopenia.
Samengo G, Avik A, Fedor B, Whittaker D, Myung KH, Wehling-Henricks M, Tidball JG.
Samengo G, et al. Among authors: myung kh.
Aging Cell. 2012 Dec;11(6):1036-45. doi: 10.1111/acel.12003. Epub 2012 Oct 4.
Aging Cell. 2012.
PMID: 22950758
Free PMC article.
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Loss of positive allosteric interactions between neuronal nitric oxide synthase and phosphofructokinase contributes to defects in glycolysis and increased fatigability in muscular dystrophy.
Wehling-Henricks M, Oltmann M, Rinaldi C, Myung KH, Tidball JG.
Wehling-Henricks M, et al. Among authors: myung kh.
Hum Mol Genet. 2009 Sep 15;18(18):3439-51. doi: 10.1093/hmg/ddp288. Epub 2009 Jun 19.
Hum Mol Genet. 2009.
PMID: 19542095
Free PMC article.
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