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Year | Number of Results |
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2009 | 2 |
2011 | 1 |
2016 | 1 |
2019 | 1 |
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Page 1
A new action of peptide hormones for survival in a low-nutrient environment.
Endocr J. 2019 Nov 28;66(11):943-952. doi: 10.1507/endocrj.EJ19-0274. Epub 2019 Sep 27.
Endocr J. 2019.
PMID: 31564683
Free article.
Review.
Physiological Evaluation of Childcare-Associated Muscle Load on the Neck and Shoulder Region in Japanese Women.
Yoshinaga S, Kiyokawa T, Kuramoto E, Kinoshita H, Nemoto S.
Yoshinaga S, et al.
Nurs Res Pract. 2016;2016:1757094. doi: 10.1155/2016/1757094. Epub 2016 Mar 15.
Nurs Res Pract. 2016.
PMID: 27064421
Free PMC article.
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Transcriptional up-regulation of cell surface Na V 1.7 sodium channels by insulin-like growth factor-1 via inhibition of glycogen synthase kinase-3β in adrenal chromaffin cells: enhancement of 22Na+ influx, 45Ca2+ influx and catecholamine secretion.
Yanagita T, Satoh S, Uezono Y, Matsuo K, Nemoto T, Maruta T, Yoshikawa N, Iwakiri T, Minami K, Murakami M.
Yanagita T, et al.
Neuropharmacology. 2011 Dec;61(8):1265-74. doi: 10.1016/j.neuropharm.2011.07.029. Epub 2011 Jul 28.
Neuropharmacology. 2011.
PMID: 21816165
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Nav1.7 sodium channel-induced Ca2+ influx decreases tau phosphorylation via glycogen synthase kinase-3beta in adrenal chromaffin cells.
Kanai T, Nemoto T, Yanagita T, Maruta T, Satoh S, Yoshikawa N, Wada A.
Kanai T, et al.
Neurochem Int. 2009 Jul;54(8):497-505. doi: 10.1016/j.neuint.2009.02.002. Epub 2009 Feb 24.
Neurochem Int. 2009.
PMID: 19428794
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Chronic lithium treatment up-regulates cell surface Na(V)1.7 sodium channels via inhibition of glycogen synthase kinase-3 in adrenal chromaffin cells: enhancement of Na(+) influx, Ca(2+) influx and catecholamine secretion after lithium withdrawal.
Yanagita T, Maruta T, Nemoto T, Uezono Y, Matsuo K, Satoh S, Yoshikawa N, Kanai T, Kobayashi H, Wada A.
Yanagita T, et al.
Neuropharmacology. 2009 Sep;57(3):311-21. doi: 10.1016/j.neuropharm.2009.05.006. Epub 2009 May 30.
Neuropharmacology. 2009.
PMID: 19486905
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