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1979 1
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Regional brain abnormalities in norepinephrine uptake and dopamine beta-hydroxylase activity in the genetically epilepsy-prone rat.
Browning RA, Wade DR, Marcinczyk M, Long GL, Jobe PC. Browning RA, et al. Among authors: wade dr. J Pharmacol Exp Ther. 1989 Apr;249(1):229-35. J Pharmacol Exp Ther. 1989. PMID: 2709332
These studies were designed to characterize further the nature of the noradrenergic deficit in GEPRs, which has been described as a reduction in steady-state NE levels. The high affinity (desmethylimipramine-sensitive) uptake of 3H-NE into crude synaptosomes was found to b …
These studies were designed to characterize further the nature of the noradrenergic deficit in GEPRs, which has been described as a reductio …
Warfarin and warfarin-alcohol levels in anticoagulated patients.
Duffield P, Duffield AM, Kennedy M, Birkett DJ, Wade DN. Duffield P, et al. Among authors: wade dn. Aust N Z J Med. 1979 Oct;9(5):534-7. doi: 10.1111/j.1445-5994.1979.tb03390.x. Aust N Z J Med. 1979. PMID: 294908
The relationship between dosage, prothrombin ratio, and steady-state plasma concentrations of warfarin and warfarin-alcohol, were determined in 43 patients regularly attending an anticoagulant clinic. ...There was no significant correlation between the prothrombin ratio an …
The relationship between dosage, prothrombin ratio, and steady-state plasma concentrations of warfarin and warfarin-alcohol, were det …
The effect of concurrent aspirin upon plasma concentrations of tenoxicam.
Day RO, Paull PD, Lam S, Swanson BR, Williams KM, Wade DN. Day RO, et al. Among authors: wade dn. Br J Clin Pharmacol. 1988 Oct;26(4):455-62. doi: 10.1111/j.1365-2125.1988.tb03405.x. Br J Clin Pharmacol. 1988. PMID: 3190995 Free PMC article.
Aspirin caused a 24% drop in the t1/2 (P less than 0.005), a 49% rise in the volume of distribution (P less than 0.0003) and a 98% increase in the clearance (P less than 0.0001) of tenoxicam after a single dose of the tenoxicam. 3. Steady-state concentrations of tenoxicam …
Aspirin caused a 24% drop in the t1/2 (P less than 0.005), a 49% rise in the volume of distribution (P less than 0.0003) and a 98% increase …
Persistent metabolic sequelae of severe head injury in humans in vivo.
Cadoux-Hudson TA, Wade D, Taylor DJ, Rajagopalan B, Ledingham JG, Briggs M, Radda GK. Cadoux-Hudson TA, et al. Among authors: wade d. Acta Neurochir (Wien). 1990;104(1-2):1-7. doi: 10.1007/BF01842884. Acta Neurochir (Wien). 1990. PMID: 2386083
The reduction in pHi so long after injury is difficult to explain in these steady-state conditions. A structural abnormality, such as a disorder in the blood brain barrier or accumulation of large acidic lysosomes, could cause these pHi changes. ...
The reduction in pHi so long after injury is difficult to explain in these steady-state conditions. A structural abnormality, such as …