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Showing results for nicolas senile
Your search for Nicolas Senil retrieved no results
Microglia in Alzheimer's Disease in the Context of Tau Pathology.
Perea JR, Bolós M, Avila J. Perea JR, et al. Biomolecules. 2020 Oct 14;10(10):1439. doi: 10.3390/biom10101439. Biomolecules. 2020. PMID: 33066368 Free PMC article. Review.
This pathology is characterized by the accumulation of amyloid-beta peptide (Abeta), which forms senile plaques in the neocortex, as well as by the aggregation of hyperphosphorylated tau protein, a process that leads to the development of neurofibrillary tangles (NFTs). .. …
This pathology is characterized by the accumulation of amyloid-beta peptide (Abeta), which forms senile plaques in the neocortex, as …
Phenotype and imaging features associated with APP duplications.
Grangeon L, Charbonnier C, Zarea A, Rousseau S, Rovelet-Lecrux A, Bendetowicz D, Lemaitre M, Malrain C, Quillard-Muraine M, Cassinari K, Maltete D, Pariente J, Moreaud O, Magnin E, Cretin B, Mackowiak MA, Sillaire AR, Vercelletto M, Dionet E, Felician O, Rod-Olivieri P, Thomas-Antérion C, Godeneche G, Sauvée M, Cartz-Piver L, Le Ber I, Chauvire V, Jonveaux T, Balageas AC, Laquerriere A, Duyckaerts C, Vital A, de Paula AM, Meyronet D, Guyant-Marechal L, Hannequin D, Tournier-Lasserve E, Campion D; CNR-MAJ collaborators; Nicolas G, Wallon D. Grangeon L, et al. Alzheimers Res Ther. 2023 May 11;15(1):93. doi: 10.1186/s13195-023-01172-2. Alzheimers Res Ther. 2023. PMID: 37170141 Free PMC article.
Deep microbleeds were found in two APP duplication carriers. In addition to neurofibrillary tangles and senile plaques, CAA was diffuse and severe with thickening of leptomeningeal vessels in all 9 autopsies. ...
Deep microbleeds were found in two APP duplication carriers. In addition to neurofibrillary tangles and senile plaques, CAA was diffu …
The Madrid School of Neurology (1885-1939).
Giménez-Roldán S. Giménez-Roldán S. Rev Neurol (Paris). 2015 Jan;171(1):5-15. doi: 10.1016/j.neurol.2014.08.002. Epub 2014 Oct 13. Rev Neurol (Paris). 2015. PMID: 25444449
Luis Simarro, trained in Paris, taught many talented students including Gayarre, Achucarro and Lafora. The untimely death of Nicolas Achucarro curtailed his promising career, but he still completed the clinicopathological study of the first American case of Alzheimer's dis …
Luis Simarro, trained in Paris, taught many talented students including Gayarre, Achucarro and Lafora. The untimely death of Nicolas …
Inhibition of PMCA activity by tau as a function of aging and Alzheimer's neuropathology.
Berrocal M, Corbacho I, Vázquez-Hernández M, Ávila J, Sepúlveda MR, Mata AM. Berrocal M, et al. Biochim Biophys Acta. 2015 Jul;1852(7):1465-76. doi: 10.1016/j.bbadis.2015.04.007. Epub 2015 Apr 16. Biochim Biophys Acta. 2015. PMID: 25892185 Free article.
This disease is characterized by the accumulation of beta-amyloid senile plaques and neurofibrillary tangles of tau, a protein that plays a key role in axonal transport. ...
This disease is characterized by the accumulation of beta-amyloid senile plaques and neurofibrillary tangles of tau, a protein that p …
Epigenetic control of somatostatin and cortistatin expression by β amyloid peptide.
Rubio A, Sánchez-Mut JV, García E, Velasquez ZD, Oliver J, Esteller M, Avila J. Rubio A, et al. J Neurosci Res. 2012 Jan;90(1):13-20. doi: 10.1002/jnr.22731. Epub 2011 Sep 15. J Neurosci Res. 2012. PMID: 21922516
beta Amyloid, present in senile plaques, has been related largely to neuronal loss in the brain of patients with Alzheimer's disease. ...
beta Amyloid, present in senile plaques, has been related largely to neuronal loss in the brain of patients with Alzheimer's disease. …
Lipid peroxidation and free radical scavengers in Alzheimer's disease.
Jeandel C, Nicolas MB, Dubois F, Nabet-Belleville F, Penin F, Cuny G. Jeandel C, et al. Gerontology. 1989;35(5-6):275-82. doi: 10.1159/000213037. Gerontology. 1989. PMID: 2630382
Lipid peroxidation products and defenses against free radical damage were determined in serum of 55 patients with senile dementia of the Alzheimer type (SDAT) and compared with values in 24 age-matched healthy control subjects. ...
Lipid peroxidation products and defenses against free radical damage were determined in serum of 55 patients with senile dementia of …
Role of glycogen synthase kinase-3 in Alzheimer's disease pathogenesis and glycogen synthase kinase-3 inhibitors.
Avila J, Wandosell F, Hernández F. Avila J, et al. Expert Rev Neurother. 2010 May;10(5):703-10. doi: 10.1586/ern.10.40. Expert Rev Neurother. 2010. PMID: 20420491
Glycogen synthase kinase (GSK)-3 has been proposed as the link between the two histopathological hallmarks of Alzheimer's disease, the extracellular senile plaques composed of beta-amyloid and the intracellular neurofibrillary tangles formed from hyperphosphorylated tau. . …
Glycogen synthase kinase (GSK)-3 has been proposed as the link between the two histopathological hallmarks of Alzheimer's disease, the extra …
Nogo-A expression in the human hippocampus in normal aging and in Alzheimer disease.
Gil V, Nicolas O, Mingorance A, Ureña JM, Tang BL, Hirata T, Sáez-Valero J, Ferrer I, Soriano E, del Río JA. Gil V, et al. J Neuropathol Exp Neurol. 2006 May;65(5):433-44. doi: 10.1097/01.jnen.0000222894.59293.98. J Neuropathol Exp Neurol. 2006. PMID: 16772867
Interestingly, Nogo-A is over-expressed by hippocampal neurons in AD and is associated with beta-amyloid deposits in senile plaques. Taken together, our results reinforce the hypothesis that Reticulon proteins such as Nogo-A participate in the neuronal responses stemming f …
Interestingly, Nogo-A is over-expressed by hippocampal neurons in AD and is associated with beta-amyloid deposits in senile plaques. …