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Year | Number of Results |
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2017 | 2 |
2018 | 1 |
2021 | 2 |
2022 | 1 |
2024 | 0 |
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Page 1
Tumor factors stimulate lysosomal degradation of tumor antigens and undermine their cross-presentation in lung cancer.
Nat Commun. 2022 Nov 4;13(1):6623. doi: 10.1038/s41467-022-34428-w.
Nat Commun. 2022.
PMID: 36333297
Free PMC article.
Chemotherapy-Exacerbated Breast Cancer Metastasis: A Paradox Explainable by Dysregulated Adaptive-Response.
Middleton JD, Stover DG, Hai T.
Middleton JD, et al.
Int J Mol Sci. 2018 Oct 26;19(11):3333. doi: 10.3390/ijms19113333.
Int J Mol Sci. 2018.
PMID: 30373101
Free PMC article.
Review.
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Stress-Inducible Gene Atf3 Dictates a Dichotomous Macrophage Activity in Chemotherapy-Enhanced Lung Colonization.
Middleton JD, Fehlman J, Sivakumar S, Stover DG, Hai T.
Middleton JD, et al.
Int J Mol Sci. 2021 Jul 8;22(14):7356. doi: 10.3390/ijms22147356.
Int J Mol Sci. 2021.
PMID: 34298975
Free PMC article.
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Chemotherapy-Induced Changes in the Lung Microenvironment: The Role of MMP-2 in Facilitating Intravascular Arrest of Breast Cancer Cells.
Middleton JD, Sivakumar S, Hai T.
Middleton JD, et al.
Int J Mol Sci. 2021 Sep 24;22(19):10280. doi: 10.3390/ijms221910280.
Int J Mol Sci. 2021.
PMID: 34638621
Free PMC article.
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Stress-inducible gene Atf3 in the noncancer host cells contributes to chemotherapy-exacerbated breast cancer metastasis.
Chang YS, Jalgaonkar SP, Middleton JD, Hai T.
Chang YS, et al. Among authors: middleton jd.
Proc Natl Acad Sci U S A. 2017 Aug 22;114(34):E7159-E7168. doi: 10.1073/pnas.1700455114. Epub 2017 Aug 7.
Proc Natl Acad Sci U S A. 2017.
PMID: 28784776
Free PMC article.
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PDGFR-modulated miR-23b cluster and miR-125a-5p suppress lung tumorigenesis by targeting multiple components of KRAS and NF-kB pathways.
Naidu S, Shi L, Magee P, Middleton JD, Laganá A, Sahoo S, Leong HS, Galvin M, Frese K, Dive C, Guzzardo V, Fassan M, Garofalo M.
Naidu S, et al. Among authors: middleton jd.
Sci Rep. 2017 Nov 13;7(1):15441. doi: 10.1038/s41598-017-14843-6.
Sci Rep. 2017.
PMID: 29133857
Free PMC article.
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