Skip to main page content
U.S. flag

An official website of the United States government

Dot gov

The .gov means it’s official.
Federal government websites often end in .gov or .mil. Before sharing sensitive information, make sure you’re on a federal government site.

Https

The site is secure.
The https:// ensures that you are connecting to the official website and that any information you provide is encrypted and transmitted securely.

Access keys NCBI Homepage MyNCBI Homepage Main Content Main Navigation

Search Page

Filters

My NCBI Filters

Results by year

Table representation of search results timeline featuring number of search results per year.

Year Number of Results
2006 1
2015 1
2017 1
2018 2
2024 0

Text availability

Article attribute

Article type

Publication date

Search Results

4 results

Results by year

Filters applied: . Clear all
Page 1
Showing results for jeff camkii
Your search for Jeff Camkin retrieved no results
CaMKII in Vascular Signalling: "Friend or Foe"?
Ebenebe OV, Heather A, Erickson JR. Ebenebe OV, et al. Heart Lung Circ. 2018 May;27(5):560-567. doi: 10.1016/j.hlc.2017.12.003. Epub 2017 Dec 19. Heart Lung Circ. 2018. PMID: 29409723 Review.
For example, CaMKII is known to have a dual role in cardiac physiology and pathology. ...This review discusses the evidence for CaMKII signalling within the vasculature, with the aim to better understand both positive and potentially deleterious effects of CaMKII
For example, CaMKII is known to have a dual role in cardiac physiology and pathology. ...This review discusses the evidence for Ca
Dichotomy of Ca2+ in the heart: contraction versus intracellular signaling.
Molkentin JD. Molkentin JD. J Clin Invest. 2006 Mar;116(3):623-6. doi: 10.1172/JCI27824. J Clin Invest. 2006. PMID: 16511595 Free PMC article. Review.
One of the remaining questions in cardiac biology is how Ca2+-dependent signaling pathways are regulated under conditions of continual Ca2+ transients that mediate cardiac contraction during each heartbeat. Ca2+-calmodulin-dependent protein kinase II (CaMKII) activation an …
One of the remaining questions in cardiac biology is how Ca2+-dependent signaling pathways are regulated under conditions of continual Ca2+ …
Inhibition of calcium/calmodulin-dependent kinase II restores contraction and relaxation in isolated cardiac muscle from type 2 diabetic rats.
Daniels LJ, Wallace RS, Nicholson OM, Wilson GA, McDonald FJ, Jones PP, Baldi JC, Lamberts RR, Erickson JR. Daniels LJ, et al. Cardiovasc Diabetol. 2018 Jun 14;17(1):89. doi: 10.1186/s12933-018-0732-x. Cardiovasc Diabetol. 2018. PMID: 29903013 Free PMC article.
Basal cardiac contractility and relaxation were impaired in the cardiac muscles from the diabetic rats, and CaMKII inhibition with KN93 partially restored contractility and relaxation. Autocamtide-2-related-inhibitor peptide (AIP), another CaMKII inhibitor that acts …
Basal cardiac contractility and relaxation were impaired in the cardiac muscles from the diabetic rats, and CaMKII inhibition with KN …
STIM1 elevation in the heart results in aberrant Ca²⁺ handling and cardiomyopathy.
Correll RN, Goonasekera SA, van Berlo JH, Burr AR, Accornero F, Zhang H, Makarewich CA, York AJ, Sargent MA, Chen X, Houser SR, Molkentin JD. Correll RN, et al. J Mol Cell Cardiol. 2015 Oct;87:38-47. doi: 10.1016/j.yjmcc.2015.07.032. Epub 2015 Aug 1. J Mol Cell Cardiol. 2015. PMID: 26241845 Free PMC article.
Associated with this enhanced Ca(2+) profile was an increase in cardiac nuclear factor of activated T-cells (NFAT) and Ca(2+)/calmodulin-dependent kinase II (CaMKII) activity. We conclude that STIM1 has an unexpected function in the heart where it alters communication betw …
Associated with this enhanced Ca(2+) profile was an increase in cardiac nuclear factor of activated T-cells (NFAT) and Ca(2+)/calmodulin-dep …