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Year | Number of Results |
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Page 1
WDR62 mediates TNFα-dependent JNK activation via TRAF2-MLK3 axis.
Mol Biol Cell. 2018 Oct 1;29(20):2470-2480. doi: 10.1091/mbc.E17-08-0504. Epub 2018 Aug 9.
Mol Biol Cell. 2018.
PMID: 30091641
Free PMC article.
Breast Cancer-Derived Microparticles Reduce Cancer Cell Adhesion, an Effect Augmented by Chemotherapy.
Shechter D, Harel M, Mukherjee A, Sagredo LM, Loven D, Prinz E, Avraham S, Orian-Rousseau V, Geiger T, Shaked Y, Wolfenson H.
Shechter D, et al. Among authors: prinz e.
Cells. 2020 Oct 10;9(10):2269. doi: 10.3390/cells9102269.
Cells. 2020.
PMID: 33050539
Free PMC article.
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Intratumor Heterogeneity of KRAS Mutation Status in Pancreatic Ductal Adenocarcinoma Is Associated With Smaller Lesions.
Nagawkar SS, Abu-Funni S, Simon E, Bick T, Prinz E, Sabo E, Ben-Izhak O, Hershkovitz D.
Nagawkar SS, et al. Among authors: prinz e.
Pancreas. 2016 Jul;45(6):876-81. doi: 10.1097/MPA.0000000000000562.
Pancreas. 2016.
PMID: 26646269
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Subclonality for BRAF Mutation in Papillary Thyroid Carcinoma Is Associated With Earlier Disease Stage.
Finkel A, Liba L, Simon E, Bick T, Prinz E, Sabo E, Ben-Izhak O, Hershkovitz D.
Finkel A, et al. Among authors: prinz e.
J Clin Endocrinol Metab. 2016 Apr;101(4):1407-13. doi: 10.1210/jc.2015-4031. Epub 2016 Feb 2.
J Clin Endocrinol Metab. 2016.
PMID: 26835544
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Clinically significant sub-clonality for common drivers can be detected in 26% of KRAS/EGFR mutated lung adenocarcinomas.
Simon E, Bick T, Sarji S, Shentzer T, Prinz E, Yehiam L, Sabo E, Ben-Izhak O, Hershkovitz D.
Simon E, et al. Among authors: prinz e.
Oncotarget. 2017 Jul 11;8(28):45736-45749. doi: 10.18632/oncotarget.17399.
Oncotarget. 2017.
PMID: 28501852
Free PMC article.
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Adenoma and carcinoma components in colonic tumors show discordance for KRAS mutation.
Hershkovitz D, Simon E, Bick T, Prinz E, Noy S, Sabo E, Ben-Izhak O, Vieth M.
Hershkovitz D, et al. Among authors: prinz e.
Hum Pathol. 2014 Sep;45(9):1866-71. doi: 10.1016/j.humpath.2014.05.005. Epub 2014 May 28.
Hum Pathol. 2014.
PMID: 24998492
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Variation in KRAS driver substitution distributions between tumor types is determined by both mutation and natural selection.
Ostrow SL, Simon E, Prinz E, Bick T, Shentzer T, Nagawkar SS, Sabo E, Ben-Izhak O, Hershberg R, Hershkovitz D.
Ostrow SL, et al. Among authors: prinz e.
Sci Rep. 2016 Feb 23;6:21927. doi: 10.1038/srep21927.
Sci Rep. 2016.
PMID: 26902163
Free PMC article.
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