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Page 1
Hypothesis and Theory: Roles of Arginine Methylation in C9orf72-Mediated ALS and FTD.
Front Cell Neurosci. 2021 Mar 23;15:633668. doi: 10.3389/fncel.2021.633668. eCollection 2021.
Front Cell Neurosci. 2021.
PMID: 33833668
Free PMC article.
Type I PRMT Inhibition Protects Against C9ORF72 Arginine-Rich Dipeptide Repeat Toxicity.
Premasiri AS, Gill AL, Vieira FG.
Premasiri AS, et al. Among authors: gill al.
Front Pharmacol. 2020 Sep 8;11:569661. doi: 10.3389/fphar.2020.569661. eCollection 2020.
Front Pharmacol. 2020.
PMID: 33013410
Free PMC article.
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Reduced C9orf72 expression exacerbates polyGR toxicity in patient iPSC-derived motor neurons and a Type I protein arginine methyltransferase inhibitor reduces that toxicity.
Dane TL, Gill AL, Vieira FG, Denton KR.
Dane TL, et al. Among authors: gill al.
Front Cell Neurosci. 2023 Apr 17;17:1134090. doi: 10.3389/fncel.2023.1134090. eCollection 2023.
Front Cell Neurosci. 2023.
PMID: 37138766
Free PMC article.
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Primary Neurons and Differentiated NSC-34 Cells Are More Susceptible to Arginine-Rich ALS Dipeptide Repeat Protein-Associated Toxicity than Non-Differentiated NSC-34 and CHO Cells.
Gill AL, Wang MZ, Levine B, Premasiri A, Vieira FG.
Gill AL, et al.
Int J Mol Sci. 2019 Dec 11;20(24):6238. doi: 10.3390/ijms20246238.
Int J Mol Sci. 2019.
PMID: 31835664
Free PMC article.
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