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Impairment of Endothelial Function by Cigarette Smoke Is Not Caused by a Specific Smoke Constituent, but by Vagal Input From the Airway.
Nabavizadeh P, Liu J, Rao P, Ibrahim S, Han DD, Derakhshandeh R, Qiu H, Wang X, Glantz SA, Schick SF, Springer ML. Nabavizadeh P, et al. Among authors: wang x. Arterioscler Thromb Vasc Biol. 2022 Nov;42(11):1324-1332. doi: 10.1161/ATVBAHA.122.318051. Epub 2022 Oct 26. Arterioscler Thromb Vasc Biol. 2022. PMID: 36288292 Free PMC article.
Donor myocardial infarction impairs the therapeutic potential of bone marrow cells by an interleukin-1-mediated inflammatory response.
Wang X, Takagawa J, Lam VC, Haddad DJ, Tobler DL, Mok PY, Zhang Y, Clifford BT, Pinnamaneni K, Saini SA, Su R, Bartel MJ, Sievers RE, Carbone L, Kogan S, Yeghiazarians Y, Hermiston M, Springer ML. Wang X, et al. Sci Transl Med. 2011 Sep 14;3(100):100ra90. doi: 10.1126/scitranslmed.3002814. Sci Transl Med. 2011. PMID: 21918107 Free PMC article.
Brief exposure to secondhand smoke reversibly impairs endothelial vasodilatory function.
Pinnamaneni K, Sievers RE, Sharma R, Selchau AM, Gutierrez G, Nordsieck EJ, Su R, An S, Chen Q, Wang X, Derakhshandeh R, Aschbacher K, Heiss C, Glantz SA, Schick SF, Springer ML. Pinnamaneni K, et al. Among authors: wang x. Nicotine Tob Res. 2014 May;16(5):584-90. doi: 10.1093/ntr/ntt189. Epub 2013 Dec 3. Nicotine Tob Res. 2014. PMID: 24302638 Free PMC article.
Overexpression of Nitric Oxide Synthase Restores Circulating Angiogenic Cell Function in Patients With Coronary Artery Disease: Implications for Autologous Cell Therapy for Myocardial Infarction.
Chen Q, Varga M, Wang X, Haddad DJ, An S, Medzikovic L, Derakhshandeh R, Kostyushev DS, Zhang Y, Clifford BT, Luu E, Danforth OM, Rafikov R, Gong W, Black SM, Suchkov SV, Fineman JR, Heiss C, Aschbacher K, Yeghiazarians Y, Springer ML. Chen Q, et al. Among authors: wang x. J Am Heart Assoc. 2016 Jan 6;5(1):e002257. doi: 10.1161/JAHA.115.002257. J Am Heart Assoc. 2016. PMID: 26738788 Free PMC article.
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