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JNK-Dependent cJun Phosphorylation Mitigates TGFβ- and EGF-Induced Pre-Malignant Breast Cancer Cell Invasion by Suppressing AP-1-Mediated Transcriptional Responses.
Cells. 2019 Nov 21;8(12):1481. doi: 10.3390/cells8121481.
Cells. 2019.
PMID: 31766464
Free PMC article.
ATF3 and Fra1 have opposite functions in JNK- and ERK-dependent DNA damage responses.
Hamdi M, Popeijus HE, Carlotti F, Janssen JM, van der Burgt C, Cornelissen-Steijger P, van de Water B, Hoeben RC, Matsuo K, van Dam H.
Hamdi M, et al.
DNA Repair (Amst). 2008 Mar 1;7(3):487-96. doi: 10.1016/j.dnarep.2007.12.004. Epub 2008 Jan 30.
DNA Repair (Amst). 2008.
PMID: 18249159
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DNA damage in transcribed genes induces apoptosis via the JNK pathway and the JNK-phosphatase MKP-1.
Hamdi M, Kool J, Cornelissen-Steijger P, Carlotti F, Popeijus HE, van der Burgt C, Janssen JM, Yasui A, Hoeben RC, Terleth C, Mullenders LH, van Dam H.
Hamdi M, et al.
Oncogene. 2005 Nov 3;24(48):7135-44. doi: 10.1038/sj.onc.1208875.
Oncogene. 2005.
PMID: 16044158
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Growth factors can activate ATF2 via a two-step mechanism: phosphorylation of Thr71 through the Ras-MEK-ERK pathway and of Thr69 through RalGDS-Src-p38.
Ouwens DM, de Ruiter ND, van der Zon GC, Carter AP, Schouten J, van der Burgt C, Kooistra K, Bos JL, Maassen JA, van Dam H.
Ouwens DM, et al.
EMBO J. 2002 Jul 15;21(14):3782-93. doi: 10.1093/emboj/cdf361.
EMBO J. 2002.
PMID: 12110590
Free PMC article.
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