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Differentially Expressed mRNA Targets of Differentially Expressed miRNAs Predict Changes in the TP53 Axis and Carcinogenesis-Related Pathways in Human Keratinocytes Chronically Exposed to Arsenic.
Toxicol Sci. 2018 Apr 1;162(2):645-654. doi: 10.1093/toxsci/kfx292.
Toxicol Sci. 2018.
PMID: 29319823
Free PMC article.
Arsenic exposure disrupts the normal function of the FA/BRCA repair pathway.
Peremartí J, Ramos F, Marcos R, Hernández A.
Peremartí J, et al.
Toxicol Sci. 2014 Nov;142(1):93-104. doi: 10.1093/toxsci/kfu159. Epub 2014 Aug 4.
Toxicol Sci. 2014.
PMID: 25092648
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Oxidative DNA damage enhances the carcinogenic potential of in vitro chronic arsenic exposures.
Bach J, Peremartí J, Annangi B, Marcos R, Hernández A.
Bach J, et al. Among authors: peremarti j.
Arch Toxicol. 2016 Aug;90(8):1893-905. doi: 10.1007/s00204-015-1605-7. Epub 2015 Oct 5.
Arch Toxicol. 2016.
PMID: 26438402
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Role of As3mt and Mth1 in the genotoxic and carcinogenic effects induced by long-term exposures to arsenic in MEF cells.
Barguilla I, Peremartí J, Bach J, Marcos R, Hernández A.
Barguilla I, et al. Among authors: peremarti j.
Toxicol Appl Pharmacol. 2020 Dec 15;409:115303. doi: 10.1016/j.taap.2020.115303. Epub 2020 Oct 24.
Toxicol Appl Pharmacol. 2020.
PMID: 33141059
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FRA1 is essential for the maintenance of the oncogenic phenotype induced by in vitro long-term arsenic exposure.
Barguilla I, Bach J, Peremartí J, Marcos R, Hernández A.
Barguilla I, et al. Among authors: peremarti j.
Metallomics. 2020 Dec 23;12(12):2161-2173. doi: 10.1039/d0mt00209g.
Metallomics. 2020.
PMID: 33313624
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Reduced cellular DNA repair capacity after environmentally relevant arsenic exposure. Influence of Ogg1 deficiency.
Bach J, Peremartí J, Annangi B, Marcos R, Hernández A.
Bach J, et al. Among authors: peremarti j.
Mutat Res. 2015 Sep;779:144-51. doi: 10.1016/j.mrfmmm.2015.07.004. Epub 2015 Jul 13.
Mutat Res. 2015.
PMID: 26210637
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