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Mechanism of hypertriglyceridemia in human apolipoprotein (apo) CIII transgenic mice. Diminished very low density lipoprotein fractional catabolic rate associated with increased apo CIII and reduced apo E on the particles.
Aalto-Setälä K, Fisher EA, Chen X, Chajek-Shaul T, Hayek T, Zechner R, Walsh A, Ramakrishnan R, Ginsberg HN, Breslow JL. Aalto-Setälä K, et al. Among authors: fisher ea. J Clin Invest. 1992 Nov;90(5):1889-900. doi: 10.1172/JCI116066. J Clin Invest. 1992. PMID: 1430212 Free PMC article.
Apoprotein B100 has a prolonged interaction with the translocon during which its lipidation and translocation change from dependence on the microsomal triglyceride transfer protein to independence.
Mitchell DM, Zhou M, Pariyarath R, Wang H, Aitchison JD, Ginsberg HN, Fisher EA. Mitchell DM, et al. Among authors: fisher ea. Proc Natl Acad Sci U S A. 1998 Dec 8;95(25):14733-8. doi: 10.1073/pnas.95.25.14733. Proc Natl Acad Sci U S A. 1998. PMID: 9843958 Free PMC article.
The amino-terminal domain of apolipoprotein B does not undergo retrograde translocation from the endoplasmic reticulum to the cytosol. Proteasomal degradation of nascent apolipoprotein B begins at the carboxyl terminus of the protein, while apolipoprotein B is still in its original translocon.
Liang S, Wu X, Fisher EA, Ginsberg HN. Liang S, et al. Among authors: fisher ea. J Biol Chem. 2000 Oct 13;275(41):32003-10. doi: 10.1074/jbc.M004646200. J Biol Chem. 2000. PMID: 10922368 Free article.
The late addition of core lipids to nascent apolipoprotein B100, resulting in the assembly and secretion of triglyceride-rich lipoproteins, is independent of both microsomal triglyceride transfer protein activity and new triglyceride synthesis.
Pan M, Liang Js JS, Fisher EA, Ginsberg HN. Pan M, et al. Among authors: fisher ea. J Biol Chem. 2002 Feb 8;277(6):4413-21. doi: 10.1074/jbc.M107460200. Epub 2001 Nov 9. J Biol Chem. 2002. PMID: 11704664 Free article.
457 results