Adenovirus-mediated gene transfer of A20 renders endothelial cells resistant to activation: a means of evaluating the role of endothelial cell activation in xenograft rejection

Transplant Proc. 1997 Feb-Mar;29(1-2):879-80. doi: 10.1016/s0041-1345(96)00184-4.
No abstract available

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adenoviruses, Human*
  • Animals
  • Cell Line
  • DNA-Binding Proteins / biosynthesis
  • E-Selectin / biosynthesis
  • Endothelium, Vascular / physiology*
  • Gene Expression
  • Genes, Reporter
  • Genome, Viral
  • Graft Rejection*
  • Humans
  • I-kappa B Proteins*
  • Interleukin-8 / biosynthesis
  • Intracellular Signaling Peptides and Proteins
  • NF-KappaB Inhibitor alpha
  • NF-kappa B / antagonists & inhibitors
  • Nuclear Proteins
  • Protein Biosynthesis*
  • Proteins*
  • RNA, Messenger / biosynthesis
  • Swine
  • Transcription, Genetic*
  • Transfection / methods*
  • Transplantation, Heterologous / immunology
  • Transplantation, Heterologous / physiology*
  • Tumor Necrosis Factor alpha-Induced Protein 3
  • Tumor Necrosis Factor-alpha / physiology
  • Zinc Fingers

Substances

  • DNA-Binding Proteins
  • E-Selectin
  • I-kappa B Proteins
  • Interleukin-8
  • Intracellular Signaling Peptides and Proteins
  • NF-kappa B
  • NFKBIA protein, human
  • Nuclear Proteins
  • Proteins
  • RNA, Messenger
  • Tumor Necrosis Factor-alpha
  • NF-KappaB Inhibitor alpha
  • TNFAIP3 protein, human
  • Tumor Necrosis Factor alpha-Induced Protein 3