Late acute rejection in renal allograft recipients is mediated by inflammatory rather than by cytotoxic T-cell-dependent mechanisms

Transplant Proc. 1997 Feb-Mar;29(1-2):93-5. doi: 10.1016/s0041-1345(96)00019-x.

Authors

S Ode-Hakim¹, W D Döcke, S Mutze, H D volk, P Reinke

Affiliation

¹ Department of Radiology, Charité, Humboldt-University, Berlin, Germany.

PMID: 9123168
DOI: 10.1016/s0041-1345(96)00019-x

No abstract available

Publication types

Research Support, Non-U.S. Gov't

MeSH terms

Biomarkers
Cytokines / biosynthesis*
Cytotoxicity, Immunologic
Graft Rejection / immunology*
Graft Rejection / pathology
Granzymes
Humans
Hypersensitivity, Delayed
Inflammation
Interferon-gamma / biosynthesis
Interleukins / biosynthesis
Kidney Transplantation / immunology*
Kidney Transplantation / pathology
Receptors, Interleukin-2 / biosynthesis
Serine Endopeptidases / biosynthesis
T-Lymphocytes, Cytotoxic / immunology*
T-Lymphocytes, Cytotoxic / pathology
Time Factors
Transcription, Genetic
Transplantation, Homologous
Tumor Necrosis Factor-alpha / biosynthesis

Substances

Biomarkers
Cytokines
Interleukins
Receptors, Interleukin-2
Tumor Necrosis Factor-alpha
Interferon-gamma
Granzymes
Serine Endopeptidases
GZMA protein, human