Immunohistochemical appearance of corticosteroid contact hypersensitivity reactions

Contact Dermatitis. 1994 Nov;31(5):304-7. doi: 10.1111/j.1600-0536.1994.tb02024.x.

Abstract

We have studied, immunohistochemically, hypersensitivity reactions to corticosteroids and compared them with allergic contact dermatitis from nickel and appropriate controls. We could find no qualitative differences between nickel and corticosteroid contact reactions, providing further evidence that hypersensitivity to corticosteroids is an immunologically mediated reaction.

Publication types

  • Comparative Study

MeSH terms

  • Administration, Topical
  • Anti-Inflammatory Agents / adverse effects*
  • Cell Adhesion Molecules / genetics
  • Cell Adhesion Molecules / metabolism*
  • Dermatitis, Allergic Contact / etiology*
  • Dermatitis, Allergic Contact / metabolism
  • Dermatitis, Allergic Contact / pathology*
  • E-Selectin
  • Endothelium / metabolism
  • Endothelium / pathology
  • Gene Expression
  • Humans
  • Hydrocortisone / adverse effects*
  • Hydrocortisone / analogs & derivatives*
  • Immunohistochemistry
  • Intercellular Adhesion Molecule-1 / genetics
  • Intercellular Adhesion Molecule-1 / metabolism
  • Irritants / adverse effects
  • Keratinocytes / metabolism
  • Keratinocytes / pathology
  • Lymphocytes / pathology
  • Membrane Glycoproteins / genetics
  • Membrane Glycoproteins / metabolism
  • Nickel / adverse effects
  • Receptors, Immunologic / genetics
  • Receptors, Immunologic / metabolism

Substances

  • Anti-Inflammatory Agents
  • Cell Adhesion Molecules
  • E-Selectin
  • Irritants
  • Membrane Glycoproteins
  • Receptors, Immunologic
  • Intercellular Adhesion Molecule-1
  • nickel sulfate
  • tixocortol pivalate
  • Nickel
  • Hydrocortisone