Medin amyloid may drive arterial aging and disease in the periphery and brain
Nat Aging
.
2023 Sep;3(9):1039-1041.
doi: 10.1038/s43587-023-00481-3.
Authors
Jillian Madine
1
,
Hannah A Davies
1
,
Raymond Q Migrino
2
,
Sanni E Ruotsalainen
3
,
Jessica Wagner
4
,
Jonas J Neher
5
6
7
Affiliations
1
Institute of Systems, Molecular and Integrative Biology, University of Liverpool, Liverpool, UK.
2
Phoenix Veterans Affairs Health Care System and University of Arizona College of Medicine-Phoenix, Phoenix, AZ, USA.
3
Institute for Molecular Medicine Finland (FIMM), HiLIFE, University of Helsinki, Helsinki, Finland.
4
German Center for Neurodegenerative Diseases (DZNE), Tübingen, Germany.
5
German Center for Neurodegenerative Diseases (DZNE), Tübingen, Germany. jonas.neher@dzne.de.
6
Department of Cellular Neurology, Hertie Institute for Clinical Brain Research, University of Tübingen, Tübingen, Germany. jonas.neher@dzne.de.
7
Metabolic Biochemistry, Biomedical Center (BMC), Faculty of Medicine, LMU Munich, Munich, Germany. jonas.neher@dzne.de.
PMID:
37620584
DOI:
10.1038/s43587-023-00481-3
No abstract available
Publication types
Research Support, N.I.H., Extramural
Research Support, Non-U.S. Gov't
Research Support, U.S. Gov't, Non-P.H.S.
MeSH terms
Amyloidogenic Proteins*
Arteries
Brain*
Head
Substances
Amyloidogenic Proteins
Grants and funding
I01 BX003767/BX/BLRD VA/United States
R21 AG075543/AG/NIA NIH HHS/United States