Abstract
Keywords:
cardiovascular diseases (CVDs); cardiovascular remodeling; drug therapy; dysfunction; genetic studies.
Grants and funding
This work was supported by the National Natural Science Foundations of China (grant numbers 31972909, 82070502, and 32171099) and the Sichuan Science and Technology Program (grant numbers 2022YFS0607, 23NSFSC1456, and 2021YJ0213).