Disarm resistance: Fungal effectors target WAK alternative splicing variant for virulence

Zunyong Liu; Yunqing Jian; Libo Shan

doi:10.1016/j.celrep.2022.111939

Disarm resistance: Fungal effectors target WAK alternative splicing variant for virulence

Cell Rep. 2023 Jan 31;42(1):111939. doi: 10.1016/j.celrep.2022.111939. Epub 2023 Jan 4.

Authors

Zunyong Liu¹, Yunqing Jian¹, Libo Shan²

Affiliations

¹ Department of Biochemistry and Biophysics, Texas A&M University, College Station, TX 77843, USA.
² Department of Biochemistry and Biophysics, Texas A&M University, College Station, TX 77843, USA. Electronic address: lshan@tamu.edu.

PMID: 36640313
DOI: 10.1016/j.celrep.2022.111939

Abstract

Molecular interactions between pathogen effectors and plant immunity underpin the arms race of disease resistance and susceptibility. In a recently published Cell Reports paper, Zuo et al. reported the mechanistic characterization of Fusarium graminearum CFEM effectors that dampen ZmWAK17-mediated defenses in maize (Zea mays).

Publication types

Comment

MeSH terms

Alternative Splicing* / genetics
Disease Resistance* / genetics
Fungal Proteins / metabolism
Plant Diseases / genetics
Plant Diseases / microbiology
Virulence / genetics
Zea mays / genetics
Zea mays / metabolism
Zea mays / microbiology

Substances

Fungal Proteins