Abstract
In the multi-factorial etiology of organ-site cancers by suspect human chemical carcinogens, oncogenic virus, activation of RAS, Myc and HER-2 oncogenes, inactivation of TP53, RB and APC tumor suppressor genes represent early-occurring genetic events [...].
MeSH terms
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Antineoplastic Agents* / pharmacology
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Antineoplastic Agents* / therapeutic use
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Carcinogens
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Humans
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Neoplasms* / drug therapy
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Oncogenes
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Oncogenic Viruses
Substances
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Antineoplastic Agents
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Carcinogens
Grants and funding
This research received no external funding.