The presence of arsenic in the groundwater of the densely-populated Bengal Basin evolved as a mass-poisoning agent and is a reason for the misery of millions of people living here. High-level arsenic was detected in the shallow aquifer-tube wells of the basin in the late-20th century. The redox conditions and the biogeochemical activities in the shallow aquifers support the existence of arsenic in its most toxic +3 state. The shallow aquifers are constructed by the Holocene reduced grey sands, having a lesser capacity to hold the arsenic brought from the Himalayas by the Ganga-Brahmaputra-Meghna river system. Among several other hypotheses, the reductive dissolution of arsenic bearing Fe-oxyhydroxides coupled with the microbial activities in the organic-matter-rich Holocene grey sands is believed to be the primary reason for releasing arsenic in groundwater of basinal shallow aquifers. The deep aquifers below the late Pleistocene aquifers and the Palaeo-interfluvial aquifers capped by the last glacial maximum Palaeosol generally contain arsenic-free or low-arsenic water. Ingress of arsenic into the deep aquifers from the shallow aquifers was considered to have been caused by extensive non-domestic pumping. However, studies have found that extensive pumping is unlikely to contaminate the deep aquifer water with higher levels of arsenic within decadal time scales. Irrigation-pumping may produce hydraulic barriers between the shallow and deep aquifer-groundwater and distributes arsenic in the topsoil by flushing. Significant disparities have been observed among the Bengal basinal groundwater arsenic concentrations. However, abrupt spatial variation in groundwater arsenic concentrations has been a key feature of the basin.
Keywords: Aquifers; Arsenic mobility; Bengal basin; Biogeochemistry; Groundwater arsenic; LGM Palaeosol.
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