The renin angiotensin system and the cholinergic anti-inflammatory pathway have been recently shown to modulate lung inflammation in patients with COVID-19. We will show how studies performed on this disease are starting to provide evidence that these two anti-inflammatory systems may functionally interact with each other, a mechanism that could have a more general physiological relevance than only COVID-19 infection.
Keywords: angiotensin-converting enzyme 2; inflammation; lung; nicotinic acetylcholine receptors; severe acute respiratory syndrome coronavirus 2.
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