Abstract
State-of-the-art genetic and cellular studies uniquely implicate the S. cerevisiae Pms1 endonuclease (human PMS2) and ExoI as the major components that produce and/or maintain the strand-specific nicks that precisely direct mismatch repair.
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MeSH terms
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DNA Mismatch Repair*
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Humans
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MutL Proteins
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Saccharomyces cerevisiae / genetics
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Saccharomyces cerevisiae / metabolism
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Saccharomyces cerevisiae Proteins* / genetics
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Saccharomyces cerevisiae Proteins* / metabolism
Substances
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PMS1 protein, S cerevisiae
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Saccharomyces cerevisiae Proteins
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MutL Proteins