Bilateral extensive corticospinal tract lesions in MOG antibody-associated disease

Neurology. 2020 Oct 6;95(14):648-649. doi: 10.1212/WNL.0000000000010662. Epub 2020 Aug 14.

Authors

Vincenzo Mastrangelo¹, Gian Maria Asioli², Matteo Foschi², Marina Padroni², Lucia Pavolucci², Patrizia Cenni², Pietro Querzani²

Affiliations

¹ From the Department of Biomedical and Neuromotor Sciences (V.M., G.M.A.), University of Bologna; and Neurology Unit (M.F., M.P., L.P., P.Q.) and Neuroradiology (P.C.), S. Maria delle Croci Hospital-AUSL Romagna, Ambito Di Ravenna, Italy. vincenzo.mastrangel2@studio.unibo.it.
² From the Department of Biomedical and Neuromotor Sciences (V.M., G.M.A.), University of Bologna; and Neurology Unit (M.F., M.P., L.P., P.Q.) and Neuroradiology (P.C.), S. Maria delle Croci Hospital-AUSL Romagna, Ambito Di Ravenna, Italy.

PMID: 32817179
DOI: 10.1212/WNL.0000000000010662

No abstract available

Publication types

Case Reports

MeSH terms

Autoantibodies / blood
Autoantibodies / immunology
Autoantigens / immunology
Autoimmune Diseases of the Nervous System / drug therapy
Autoimmune Diseases of the Nervous System / immunology*
Autoimmune Diseases of the Nervous System / pathology
Demyelinating Diseases / drug therapy
Demyelinating Diseases / immunology*
Demyelinating Diseases / pathology
Humans
Immunoglobulins, Intravenous / therapeutic use
Male
Middle Aged
Myelin-Oligodendrocyte Glycoprotein / immunology*
Pyramidal Tracts / pathology*

Substances

Autoantibodies
Autoantigens
Immunoglobulins, Intravenous
MOG protein, human
Myelin-Oligodendrocyte Glycoprotein