Reply to: "Interleukin-22 in acute-on-chronic liver failure: A matter of ineffective levels, receptor dysregulation or defective signalling?": The search for an optimal mouse model

J Hepatol. 2020 Oct;73(4):982-984. doi: 10.1016/j.jhep.2020.06.002. Epub 2020 Jul 18.
No abstract available

Publication types

  • Letter
  • Research Support, N.I.H., Intramural
  • Comment

MeSH terms

  • Acute-On-Chronic Liver Failure*
  • Animals
  • Disease Models, Animal
  • Interleukin-22
  • Interleukins
  • Mice
  • Signal Transduction

Substances

  • Interleukins