Abstract
Some patients with coronavirus disease 2019 (COVID-19) show abnormal changes in laboratory myocardial injury markers, suggesting that patients with myocardial injury have a higher mortality rate than those without myocardial injury. This article reviews the possible mechanism of myocardial injury in patients with COVID-19. Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) affects the patients with COVID-19 in aspects of direct infection of myocardial injury, specific binding to functional receptors on cardiomyocytes, and immune-mediated myocardial injury. During hospitalization, the monitoring of laboratory myocardial injury markers in patients of COVID-19 should be strengthened.
Copyright © 2020 Lin Li et al.
MeSH terms
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Angiotensin-Converting Enzyme 2
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Betacoronavirus*
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Biomarkers / blood
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Biomarkers / metabolism
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COVID-19
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Coronavirus Infections / blood*
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Coronavirus Infections / complications*
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Coronavirus Infections / metabolism
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Cytokines / blood
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Cytokines / immunology
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Heart Injuries / blood*
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Heart Injuries / etiology*
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Heart Injuries / metabolism
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Humans
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Inflammation Mediators / blood
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Inflammation Mediators / immunology
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Models, Cardiovascular
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Models, Immunological
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Myocytes, Cardiac / immunology
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Myocytes, Cardiac / metabolism
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Pandemics
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Peptidyl-Dipeptidase A / metabolism
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Pneumonia, Viral / blood*
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Pneumonia, Viral / complications*
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Pneumonia, Viral / metabolism
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SARS-CoV-2
Substances
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Biomarkers
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Cytokines
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Inflammation Mediators
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Peptidyl-Dipeptidase A
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ACE2 protein, human
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Angiotensin-Converting Enzyme 2