SARS-CoV-2-mediated inflammatory response in lungs: should we look at RAGE?

Inflamm Res. 2020 Jul;69(7):641-643. doi: 10.1007/s00011-020-01353-x. Epub 2020 May 5.

Authors

Armando Rojas¹, Ileana Gonzalez², Miguel A Morales³

Affiliations

¹ Biomedical Research Laboratories, Medicine Faculty, Catholic University of Maule, Talca, Chile. arojasr@ucm.cl.
² Biomedical Research Laboratories, Medicine Faculty, Catholic University of Maule, Talca, Chile.
³ Molecular and Clinical Pharmacology Program, Institute of Biomedical Sciences, University of Chile, Santiago, Chile.

No abstract available

MeSH terms

Angiotensin II / physiology
Angiotensin-Converting Enzyme 2
Betacoronavirus*
COVID-19
Coronavirus Infections / complications*
Humans
Pandemics
Peptidyl-Dipeptidase A / physiology
Pneumonia / etiology*
Pneumonia, Viral / complications*
Receptor for Advanced Glycation End Products / physiology*
Receptor, Angiotensin, Type 1 / physiology
SARS-CoV-2

Substances

Receptor for Advanced Glycation End Products
Receptor, Angiotensin, Type 1
Angiotensin II
Peptidyl-Dipeptidase A
ACE2 protein, human
Angiotensin-Converting Enzyme 2