β-Arrestin2 arrests the clearance of tau in FTLD

Proc Natl Acad Sci U S A. 2020 Mar 31;117(13):6968-6970. doi: 10.1073/pnas.2001455117. Epub 2020 Mar 18.

Author

Amantha Thathiah^{1

2

3

4}

Affiliations

¹ Department of Neurobiology, University of Pittsburgh School of Medicine, Pittsburgh, PA 15213; amantha@pitt.edu.
² Pittsburgh Institute for Neurodegenerative Diseases, Brain Institute, University of Pittsburgh, Pittsburgh, PA 15213.
³ Center for Neuroscience, University of Pittsburgh, Pittsburgh, PA 15213.
⁴ Center for Protein Conformational Diseases, Kenneth P. Dietrich School of Arts and Sciences, University of Pittsburgh, Pittsburgh, PA 15213.

No abstract available

Publication types

Research Support, N.I.H., Extramural
Comment

MeSH terms

Amyloid beta-Peptides
Frontotemporal Lobar Degeneration*
Humans
beta-Arrestin 1
beta-Arrestin 2
tau Proteins*

Substances

Amyloid beta-Peptides
beta-Arrestin 1
beta-Arrestin 2
tau Proteins

Grants and funding

R01 AG058851/AG/NIA NIH HHS/United States