Divergent Roles of Epithelium-Derived Alarmins in Notch Signaling-Deficient Skin

J Invest Dermatol. 2020 Oct;140(10):2089-2092. doi: 10.1016/j.jid.2020.02.020. Epub 2020 Mar 6.

Authors

Jonathan L Messerschmidt¹, Kaitlin E Dempsey¹, Kayla S Dillon¹, Shadmehr Demehri²

Affiliations

¹ Center for Cancer Immunology and Cutaneous Biology Research Center, Department of Dermatology and Center for Cancer Research, Massachusetts General Hospital and Harvard Medical School, Boston, Massachusetts.
² Center for Cancer Immunology and Cutaneous Biology Research Center, Department of Dermatology and Center for Cancer Research, Massachusetts General Hospital and Harvard Medical School, Boston, Massachusetts. Electronic address: sdemehri1@mgh.harvard.edu.

No abstract available

Publication types

Letter
Research Support, N.I.H., Extramural

MeSH terms

Alarmins / physiology*
Animals
CD4-Positive T-Lymphocytes / immunology
Cytokines / physiology
Epithelium / physiology
Immunoglobulin J Recombination Signal Sequence-Binding Protein / physiology
Interleukin-33 / physiology
Mice
Receptors, Notch / physiology*
Signal Transduction / physiology
Skin Physiological Phenomena*
Thymic Stromal Lymphopoietin

Substances

Alarmins
Cytokines
Il33 protein, mouse
Immunoglobulin J Recombination Signal Sequence-Binding Protein
Interleukin-33
Rbpj protein, mouse
Receptors, Notch
Thymic Stromal Lymphopoietin
TSLP protein, mouse

Grants and funding