Sodium retention by uPA-plasmin-ENaC in nephrotic syndrome-Authors reply

Acta Physiol (Oxf). 2020 Apr;228(4):e13432. doi: 10.1111/apha.13432. Epub 2020 Jan 9.
No abstract available

Publication types

  • Letter
  • Comment

MeSH terms

  • Epithelial Sodium Channels*
  • Fibrinolysin
  • Humans
  • Nephrotic Syndrome*
  • Sodium
  • Urokinase-Type Plasminogen Activator

Substances

  • Epithelial Sodium Channels
  • Sodium
  • Fibrinolysin
  • Urokinase-Type Plasminogen Activator