Publisher Correction: Neuronal deletion of Gtf2i, associated with Williams syndrome, causes behavioral and myelin alterations rescuable by a remyelinating drug
1 McGovern Institute for Brain Research and Department of Brain & Cognitive Sciences, MIT, Cambridge, MA, USA. boazba@tauex.tau.ac.il.
2 The School of Psychological Sciences, Faculty of Social Sciences, Tel Aviv University, Tel Aviv, Israel. boazba@tauex.tau.ac.il.
3 The Sagol School of Neuroscience, Tel Aviv University, Tel Aviv, Israel. boazba@tauex.tau.ac.il.
4 F.M. Kirby Neurobiology Center, Boston Children's Hospital and Department of Neurology, Harvard Medical School, Boston, MA, USA.
5 The Sagol School of Neuroscience, Tel Aviv University, Tel Aviv, Israel.
6 The School of Psychological Sciences, Faculty of Social Sciences, Tel Aviv University, Tel Aviv, Israel.
7 McGovern Institute for Brain Research and Department of Brain & Cognitive Sciences, MIT, Cambridge, MA, USA.
8 Stanley Center for Psychiatric Research, Broad Institute of MIT and Harvard, Cambridge, MA, USA.
9 Department of Neurobiology, Harvard Medical School, Boston, MA, USA.
10 Department of Reconstructive Sciences, University of Connecticut, Farmington, CT, USA.
11 F.M. Kirby Neurobiology Center, Boston Children's Hospital and Department of Neurology, Harvard Medical School, Boston, MA, USA. zhigang.he@childrens.harvard.edu.
12 McGovern Institute for Brain Research and Department of Brain & Cognitive Sciences, MIT, Cambridge, MA, USA. fengg@mit.edu.
13 Stanley Center for Psychiatric Research, Broad Institute of MIT and Harvard, Cambridge, MA, USA. fengg@mit.edu.