Prostaglandin E2, Tumor Necrosis Factor α, and Pro-opiomelanocortin Genes as Potential Mediators of Cancer Pain in Cutaneous Squamous Cell Carcinoma of Organ Transplant Recipients

JAMA Dermatol. 2017 Mar 1;153(3):350-352. doi: 10.1001/jamadermatol.2016.4775.

Authors

Sara Regina Frauenfelder¹, Sandra Nicole Freiberger¹, Jan Nico Bouwes Bavinck², Koen D Quint³, Roel Genders³, Andreas L Serra⁴, Günther F L Hofbauer¹

Affiliations

¹ Dermatologische Klinik, UniversitätsSpital Zürich, Zürich, Switzerland.
² Department of Dermatology, Leiden University Medical Center, Leiden, the Netherlands.
³ Department of Dermatology, Leiden University Medical Center, Leiden, the Netherlands3Department of Dermatology, Roosevelt Kliniek, Leiden, the Netherlands.
⁴ Epidemiology, Biostatistics, and Prevention Institute (EBPI), University of Zurich, Zurich, Switzerland5Department of Internal Medicine and Nephrology, Klinik Hirslanden, Zurich, Switzerland.

PMID: 27926761
DOI: 10.1001/jamadermatol.2016.4775

No abstract available

Publication types

Letter

MeSH terms

Biopsy
Cancer Pain / diagnosis
Cancer Pain / genetics*
Cancer Pain / metabolism
Carcinoma, Squamous Cell / genetics*
Carcinoma, Squamous Cell / metabolism
Carcinoma, Squamous Cell / pathology
Dinoprostone / genetics*
Dinoprostone / metabolism
Female
Gene Expression Regulation, Neoplastic*
Humans
Male
Organ Transplantation
Pro-Opiomelanocortin / biosynthesis
Pro-Opiomelanocortin / genetics*
RNA, Neoplasm / genetics
Skin Neoplasms / genetics*
Skin Neoplasms / metabolism
Skin Neoplasms / pathology
Transplant Recipients
Tumor Necrosis Factor-alpha / genetics*
Tumor Necrosis Factor-alpha / metabolism

Substances

RNA, Neoplasm
Tumor Necrosis Factor-alpha
Pro-Opiomelanocortin
Dinoprostone