Acute stress is not acute: sustained enhancement of glutamate release after acute stress involves readily releasable pool size and synapsin I activation

Mol Psychiatry. 2017 Sep;22(9):1226-1227. doi: 10.1038/mp.2016.175. Epub 2016 Oct 4.

Authors

L Musazzi¹, P Tornese¹, N Sala¹, M Popoli¹

Affiliation

¹ Laboratory of Neuropsychopharmacology and Functional Neurogenomics, Department of Pharmacological and Biomolecular Sciences, University of Milan, Milan, Italy.

PMID: 27698433
DOI: 10.1038/mp.2016.175

No abstract available

Publication types

Letter

MeSH terms

Acute Disease
Animals
Cerebral Cortex / metabolism
Corticosterone / blood
Electroshock / adverse effects
Glutamic Acid / metabolism*
Male
Presynaptic Terminals / drug effects
Presynaptic Terminals / metabolism
Rats
Stress, Psychological / etiology
Stress, Psychological / metabolism*
Stress, Psychological / pathology
Sucrose / pharmacology
Synapsins / metabolism*
Synaptosomes / drug effects
Synaptosomes / metabolism
Time Factors
Tritium / metabolism

Substances

Synapsins
Tritium
Glutamic Acid
Sucrose
Corticosterone