Gab2 is essential for Bcr-Abl-mediated leukemic transformation and hydronephrosis in a chronic myeloid leukemia mouse model

Leukemia. 2016 Sep;30(9):1942-5. doi: 10.1038/leu.2016.92. Epub 2016 Apr 29.
No abstract available

Publication types

  • Letter
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adaptor Proteins, Signal Transducing
  • Animals
  • Cell Transformation, Neoplastic / genetics*
  • Disease Models, Animal
  • Fusion Proteins, bcr-abl / genetics
  • Hydronephrosis / etiology
  • Leukemia, Myelogenous, Chronic, BCR-ABL Positive / genetics
  • Leukemia, Myelogenous, Chronic, BCR-ABL Positive / metabolism*
  • Leukemia, Myelogenous, Chronic, BCR-ABL Positive / pathology*
  • Mice
  • Mice, Transgenic
  • Phosphoproteins / metabolism*

Substances

  • Adaptor Proteins, Signal Transducing
  • Gab2 protein, mouse
  • Phosphoproteins
  • Fusion Proteins, bcr-abl